Evidence mapPaperPMID 41334584Full record

ArticleBioanalysis2025

Improving sensitivity and drug tolerance of assays for neutralizing anti-drug antibodies to semaglutide and native GLP-1.

Nicoline Videbæk, Louise Jørgensen, Carina de Lemos Rieper, Lone Hummelshøj, Steffan Svejgaard Petersen, Dorthe Bianca Corlin Wøldike, Lars Ole Andresen

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Article in Bioanalysis, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

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1 · What the graph read from it

What it found

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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

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3 · Its place in the literature

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4 · The record

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5 · Who and what money

Authors and funding

7 authors.

Nicoline VidebækNon-Clinical and Clinical Assay Sciences, Novo Nordisk A/S, Måløv, Denmark.
Louise JørgensenNon-Clinical and Clinical Assay Sciences, Novo Nordisk A/S, Måløv, Denmark.
Carina de Lemos RieperNon-Clinical and Clinical Assay Sciences, Novo Nordisk A/S, Måløv, Denmark.
Lone HummelshøjNon-Clinical and Clinical Assay Sciences, Novo Nordisk A/S, Måløv, Denmark.
Steffan Svejgaard PetersenNon-Clinical and Clinical Assay Sciences, Novo Nordisk A/S, Måløv, Denmark.
Dorthe Bianca Corlin WøldikeNon-Clinical and Clinical Assay Sciences, Novo Nordisk A/S, Måløv, Denmark.
Lars Ole AndresenNon-Clinical and Clinical Assay Sciences, Novo Nordisk A/S, Måløv, Denmark.

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

aimThe purpose of this work was to optimize the sensitivity and the drug tolerance of two cell-based assays for the detection of neutralizing antibodies (nAbs) to semaglutide (a GLP-1 analogue) and to endogenous GLP-1. METHODOLOGY: The two assays were developed and validated in three distinct iterations. Enhancements in sensitivity and drug tolerance were achieved through platform optimization, sample pre-treatment and the alteration of control antibodies.

resultsThe sensitivity and drug tolerance improved gradually with the different versions of the assays. For detection of nAbs to semaglutide, sensitivity was improved from 3,400 ng to 98 ng/ml antibody, and drug tolerance was improved from 2.5 nM semaglutide when detecting 3,400 ng/ml antibodies to 4.8-5.6 nM semaglutide when detecting 1,000 ng/ml antibody. For the endogenous GLP-1 assay, sensitivity was improved from 6,900 ng/ml to 46 ng/ml antibody, and drug tolerance improved from 1.0 nM semaglutide when detecting antibody concentration of 8,800 ng/ml to 2.5 nM semaglutide when detecting 1,000 ng/ml antibody.

conclusionKey factors for enhancing the sensitivity and drug tolerance of the assays included the concentration of the drug standard for receptor activation, pre-treatment of the samples, and better understanding of the binding properties of the control antibody.

Indexed as

Antibodies, NeutralizingDrug ToleranceGlucagon-Like Peptide 1Glucagon-Like PeptidesAnimalsHumansSemaglutideAntibodies, NeutralizingGlucagon-Like Peptide 1Glucagon-Like PeptidesSemaglutideAnti-drug-antibodiesglucagon-like peptide-1neutralizing antibody assayoptimizationsemaglutide

Identifiers

PMID41334584
PMCPMC12785235

What Socratic holds

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Registered trials

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.