Evidence mapPaperPMID 41335198Full record

ReviewJournal of endocrinological investigation2026

Unveiling the complexities of hypoparathyroidism: a comprehensive review of clinical manifestations, diagnosis, and novel therapies.

Filomena Cetani, Francesco Bertoldo, Marco Bononi, Mariarita Tarallo, Valentina Camozzi, Cristiana Cipriani, Andrea Palermo, Daniela Pasquali, Guido Zavatta

Abstract readReview
In one paragraph

Review in Journal of endocrinological investigation, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 2 papers.

0numbers the graph read from it
0cells of the map it votes in
2citing papers in PubMed
field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

2 citing papers in PubMed.

  1. Review
  2. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

9 authors.

Filomena CetaniDepartment of Clinical and Experimental Medicine, Unit of Endocrinology, University of Pisa, Via Paradisa, 2 , 56124, Pisa, Italy. filomena.cetani@unipi.it.ORCID https://orcid.org/0000-0003-2558-9547
Francesco BertoldoDepartment of Medicine, Emergency Medicine, Metabolic Bone Disease Unit, Department of Medicine, University of Verona, Verona, Italy.ORCID https://orcid.org/0000-0002-5175-7340
Marco BononiDAI Chirurgia Generale, Chirurgia Plastica e Ortopedia, DU Chirurgia, AOU Policlinico Umberto I, Sapienza University of Rome, Rome, Italy.ORCID http://orcid.org/0000-0003-4944-0368
Mariarita TaralloDAI Chirurgia Generale, Chirurgia Plastica e Ortopedia, DU Chirurgia, AOU Policlinico Umberto I, Sapienza University of Rome, Rome, Italy.ORCID http://orcid.org/0000-0003-3783-4749
Valentina CamozziEndocrinology Unit, Department of Medicine, Hospital-University of Padua, Padova, Italy.ORCID http://orcid.org/0000-0002-1634-951X
Cristiana CiprianiDepartment of Clinical, Internal, Anesthesiological and Cardiovascular Sciences, Sapienza University of Rome, Rome, Italy.ORCID http://orcid.org/0000-0002-0141-1202
Andrea PalermoUnit of Metabolic Bone and Thyroid Disorders, Fondazione Policlinico Universitario Campus Bio-Medico, Rome, Italy.ORCID http://orcid.org/0000-0002-1143-4926
Daniela PasqualiDepartment of Advanced Medical and Surgical Sciences, University of Campania, "Luigi Vanvitelli", Naples, Italy.ORCID http://orcid.org/0000-0002-5674-635X
Guido ZavattaDivision of Endocrinology and Diabetes Prevention and Care, IRCCS Azienda Ospedaliero-Universitaria di Bologna, Bologna, Italy.ORCID http://orcid.org/0000-0003-3657-2070

Funding

Ascendis Pharma Ascendis Pharma
6 · The paper itself

Abstract

backgroundHypoparathyroidism (HypoPT) is a rare endocrine disorder characterized by insufficient or absent secretion of parathyroid hormone (PTH), which results in hypocalcemia, hyperphosphatemia, and disruption of calcium phosphate homeostasis. Despite advances in understanding its pathophysiology and management, HypoPT remains a complex and impactful condition associated with significant morbidity, impaired quality of life, and long-term complications affecting the skeletal, renal, and neurological systems.

methodsA literature search was performed on PubMed. Articles were selected based on their relevance to the main topic of the review, with particular attention to recent studies.

resultsThis review provides a comprehensive synthesis of the current knowledge on HypoPT, addressing its epidemiology, underlying pathophysiological mechanisms, genetic and acquired etiologies, clinical manifestations, diagnostic strategies, and chronic disease-related complications. Emphasis is placed on the genetic spectrum of the disease, challenges of postsurgical management, and burden of conventional therapy, which often fails to fully restore mineral homeostasis and patient well-being. The evolving therapeutic landscape is detailed, highlighting advances from traditional calcium and active vitamin D supplementation to innovative PTH replacement strategies. Among these, palopegteriparatide and eneboparatide (phase 3 clinical trial ongoing) are reshaping treatment paradigms by enabling more physiological restoration of calcium-phosphate balance, reducing complications, and improving patient-centered outcomes, including renal function and quality of life.

conclusionsBy integrating clinical expertise with the latest research developments, this review offers an updated and holistic perspective on HypoPT management, aiming to support clinicians in delivering effective and individualized care to patients across the spectrum of disease severity.

Indexed as

HypoparathyroidismCalciumHumansParathyroid HormoneQuality of LifeCalciumParathyroid HormoneHypoparathyroidismHypoPT diagnosisNonsurgical HypoPTPostsurgical HypoPTPTH analoguesPTH replacement therapy

Identifiers

PMID41335198
PMCPMC13053346

What Socratic holds

Textmetadata
LicenceCC BY-NC-ND
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.