ArticleEuropean journal of applied physiology2026
The impact of muscle oxidative capacity on neuromuscular recovery after exercise-induced muscle damage.
Article in European journal of applied physiology, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.
What it found
Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.
The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
The trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.
Who cites it
0 citing papers in PubMed.
No citing paper in PubMed yet.
Corrections and comments
PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.
Authors and funding
7 authors.
Funding
No grant is acknowledged in the PubMed record.
Abstract
objectiveExercise-induced muscle damage (EIMD) compromises neuromuscular function and reduces exercise tolerance. Whether muscle oxidative capacity (MOC), an index of mitochondrial function, is reduced by EIMD or influences neuromuscular recovery remains unknown. PURPOSE: This study tested the hypotheses that EIMD would reduce MOC and that higher baseline (BL) MOC would be associated with faster recovery of neuromuscular efficiency (NME).
methodsFourteen healthy adults (23 ± 3 yrs; 7M/7F) completed neuromuscular testing, and a subset of nine participants (24 ± 3 yrs; 6M/3F) underwent MOC assessment. MOC was determined from the recovery kinetics of muscle oxygen consumption using near-infrared spectroscopy during repeated arterial occlusions. MOC was determined at BL and 1 H, 24 H, 48 H, and 1 week after damage. EIMD was induced by eccentric contractions until maximal voluntary contraction (MVC) was reduced by 40%. Participants returned 1 H, 24 H, 48 H, and 1 week after damage to perform intermittent isometric contractions to task failure. NME at exercise onset was determined as the ratio of MVC-normalized EMG root mean square (%) to torque output (Nm).
resultsEIMD did not reduce MOC (BL: 1.93 ± 1.39 min
conclusionThese findings suggest MOC may support neuromuscular recovery despite being unaffected by muscle damage.
Indexed as
Identifiers
41343076What Socratic holds
Registered trials
Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.