Evidence mapPaperPMID 41354708Full record

ReviewCommunications biology2025

Catabolic rewiring in cancer impacts dietary interventions.

Christos Chinopoulos

Abstract readReview
In one paragraph

Review in Communications biology, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 2 papers.

0numbers the graph read from it
0cells of the map it votes in
2citing papers in PubMed
field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

2 citing papers in PubMed.

  1. Article
  2. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

1 author.

Christos ChinopoulosDepartment of Biochemistry, Semmelweis University, Budapest, Hungary. chinopoulos.christos@semmelweis.hu.ORCID http://orcid.org/0000-0003-0183-4149

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Tumors exhibit metabolic fuel requirements resembling those of embryonic and other highly proliferative tissues, a similarity that can be exploited to combat neoplasia. However, several challenges limit the efficacy of dietary modifications in cancer therapy. This review explores how cancer cells acquire and catabolize macromolecules to harness energy and assesses the feasibility of influencing these pathways through dietary changes. Although dietary interventions modulate the energetic substrate supply to tumors only to a limited extent, they offer significant ancillary benefits by imposing hormonal states that burden cancer metabolism, enhancing immune responses, and reducing inflammation. Integrating dietary considerations into the standard of care for cancer patients represents a promising adjunct to traditional treatments, despite their dampened impact on nutrient availability for tumor energy metabolism.

Indexed as

DietEnergy MetabolismNeoplasmsAnimalsHumans

Identifiers

PMID41354708
PMCPMC12804764

What Socratic holds

Textmetadata
LicenceCC BY-NC-ND
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.