Evidence map›Paper›PMID 41360425›Full record

Observational studyJournal for immunotherapy of cancer2025

PD-L1 phenotype classification based on expression in tumor and immune cells as a potential biomarker for optimizing anti-PD-1/CTLA-4 immunotherapies in NSCLC.

Jun Miyakoshi, Tatsuya Yoshida, Yuji Uehara, Yuki Takeyasu, Masayuki Shirasawa, Akito Fukuda, Jumpei Kashima, Shogo Kumagai, Hidehito Horinouchi, Hanako Ono and 11 more

Abstract readMulticenter StudyObservational Study
In one paragraph

Observational study in Journal for immunotherapy of cancer, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 3 papers.

0numbers the graph read from it
0cells of the map it votes in
3citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

3 citing papers in PubMed.

  1. Review
  2. Article
  3. Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

21 authors.

Jun MiyakoshiDepartment of Thoracic Oncology, National Cancer Center Hospital, Chuo-ku, Tokyo, Japan.ORCID http://orcid.org/0009-0002-9229-2276
Tatsuya YoshidaDepartment of Thoracic Oncology, National Cancer Center Hospital, Chuo-ku, Tokyo, Japan tatyoshi@ncc.go.jp.ORCID http://orcid.org/0000-0003-4896-5824
Yuji UeharaDepartment of Experimental Therapeutics, National Cancer Center Hospital, Chuo-ku, Tokyo, Japan.
Yuki TakeyasuDepartment of Respiratory Medicine, Kansai Medical University Hospital, Hirakata, Japan.
Masayuki ShirasawaDepartment of Respiratory Medicine, Kitasato University Hospital, Sagamihara, Kanagawa, Japan.ORCID http://orcid.org/0000-0001-9948-3441
Akito FukudaDivision of Cancer Immunology, National Cancer Center Research Institute, Chuo-ku, Tokyo, Japan.
Jumpei KashimaDepartment of Diagnostic Pathology, National Cancer Center Hospital, Chuo-ku, Tokyo, Japan.
Shogo KumagaiDivision of Cancer Immunology, National Cancer Center Research Institute, Chuo-ku, Tokyo, Japan.
Hidehito HorinouchiDepartment of Thoracic Oncology, National Cancer Center Hospital, Chuo-ku, Tokyo, Japan.
Hanako OnoDivision of Genome Biology, National Cancer Center Research Institute, Chuo-ku, Tokyo, Japan.
Kouya ShiraishiDivision of Genome Biology, National Cancer Center Research Institute, Chuo-ku, Tokyo, Japan.
Takashi KohnoDivision of Genome Biology, National Cancer Center Research Institute, Chuo-ku, Tokyo, Japan.
Shunsuke KondoDepartment of Chemotherapy and Palliative Care, Tokyo Women's Medical University, Shinjuku-ku, Tokyo, Japan.
Yasushi GotoDepartment of Thoracic Oncology, National Cancer Center Hospital, Chuo-ku, Tokyo, Japan.
Noboru YamamotoDepartment of Experimental Therapeutics, National Cancer Center Hospital, Chuo-ku, Tokyo, Japan.ORCID http://orcid.org/0000-0002-0787-2851
Yasushi YatabeDepartment of Diagnostic Pathology, National Cancer Center Hospital, Chuo-ku, Tokyo, Japan.
Yukio HosomiDepartment of Respiratory Medicine, Tokyo Metropolitan Cancer and Infectious Diseases Center Komagome Hospital, Bunkyo-ku, Tokyo, Japan.
Takayasu KurataDepartment of Respiratory Medicine, Kansai Medical University Hospital, Hirakata, Japan.
Katsuhiko NaokiDepartment of Respiratory Medicine, Kitasato University Hospital, Sagamihara, Kanagawa, Japan.
Takuji SuzukiDepartment of Respiratory Medicine, Chiba University, Chiba, Chiba, Japan.
Yuichiro OheDepartment of Thoracic Oncology, National Cancer Center Hospital, Chuo-ku, Tokyo, Japan.

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

backgroundCompared with conventional chemotherapy, pembrolizumab-based chemoimmunotherapy (Pembro) and nivolumab plus ipilimumab with or without two cycles of platinum-doublet chemotherapy (Nivo+Ipi) improve survival in advanced non-small cell lung cancer (NSCLC). However, biomarkers for selecting optimal immunotherapy remain unclear. This study aimed to assess whether programmed cell death-ligand 1 (PD-L1) expression on tumor and immune cells (ICs) can guide first-line immunotherapy in advanced NSCLC.

methodsThis multicenter, observational study retrospectively reviewed patients with NSCLC treated with first-line Pembro or Nivo+Ipi who had evaluable PD-L1 expression on tumor (Tumor Proportion Score (TPS), 22C3) and ICs score (SP142). In addition, whole-exome and RNA sequencing were performed on treatment-naïve NSCLCs with available PD-L1 expression status by both assays.

resultsBetween 2019 and 2023, 198 patients were included (Pembro/Nivo+Ipi: 137/61). In the Pembro cohort, patients with high TPS (≥ 50%) had significantly longer progression-free survival (PFS) than those with low TPS (< 50%) (median PFS (mPFS, months): 8.1 vs 7.1; p=0.02), while IC score was not predictive. In the Nivo+Ipi cohort, high IC score (≥1) was associated with longer PFS than low IC score (0) (mPFS: 7.7 vs 2.8; p=0.04), while TPS showed no impact. Among patients with low TPS/high IC scores, Nivo+Ipi achieved longer PFS than Pembro (mPFS: 12.4 vs 6.6; restricted mean survival time (RMST)

conclusionsPatients with NSCLC and low TPS/high IC scores may benefit more from Nivo+Ipi than from Pembro due to distinct genomic and immunological features, including high TMB and T

Indexed as

B7-H1 AntigenBiomarkers, TumorCarcinoma, Non-Small-Cell LungCTLA-4 AntigenImmune Checkpoint InhibitorsImmunotherapyLung NeoplasmsAdultAgedAged, 80 and overFemaleHumansMaleMiddle AgedPhenotypeRetrospective StudiesB7-H1 AntigenBiomarkers, TumorCD274 protein, humanCTLA-4 AntigenCTLA4 protein, humanImmune Checkpoint InhibitorsBiomarkerImmune Checkpoint InhibitorLung CancerTumor microenvironment - TMETumor mutation burden - TMB

Identifiers

PMID41360425
PMCPMC12684186

What Socratic holds

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LicenceCC BY-NC
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Registered trials

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.