Evidence mapPaperPMID 41400708Full record

ReviewCurrent cardiology reports2025

Preserving the Metabolic Engine: Muscle as the Therapeutic Target for Cardiovascular Prevention in Obesity Pharmacotherapy.

Fabian Sanchis-Gomar, Ian J Neeland, Pilar Ruiz-Lozano, Osama Alnahar, Fatima Rodriguez

Abstract readReview
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In one paragraph

Review in Current cardiology reports, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

0numbers the graph read from it
0cells of the map it votes in
0citing papers in PubMed
field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

0 citing papers in PubMed.

No citing paper in PubMed yet.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

5 authors.

Fabian Sanchis-GomarDepartment of Medicine, Division of Cardiovascular Medicine, Stanford University School of Medicine, Stanford, CA, USA. sangofa@stanford.edu.
Ian J NeelandDepartment of Medicine, Case Western Reserve University School of Medicine, Cleveland, OH, USA.
Pilar Ruiz-LozanoRegencor, Inc., 733 Industrial Rd, CA, 94070, San Carlos, USA.
Osama AlnaharDepartment of Medicine, Division of Cardiovascular Medicine, Stanford University School of Medicine, Stanford, CA, USA.
Fatima RodriguezDepartment of Medicine, Division of Cardiovascular Medicine, Stanford University School of Medicine, Stanford, CA, USA.

Funding

Adherence Determinants in the Health Electronic Record Evaluation of Statins (ADHERES)R01HL168188 · STANFORD UNIVERSITY · 2025 to 2025
$685k
Adipose Dysfunction, Imaging, Physiology, and Outcomes with SGLT2i's for Sleep Apnea: The ADIPOSA StudyR01HL163627 · CASE WESTERN RESERVE UNIVERSITY · 2025 to 2025
$636k
NHLBI NIH HHS R01 HL163627NHLBI NIH HHS R01HL168188
6 · The paper itself

Abstract

purpose of reviewThis review examines the impact of incretin-based therapies and related incretin therapies on skeletal muscle health during pharmacologic weight loss. It explores the extent to which lean mass reduction contributes to total weight loss and highlights strategies to preserve muscle as a determinant of cardiovascular resilience. RECENT

findingsEmerging data indicate that 25-40% of incretin-based therapies-induced weight loss derives from loss of lean mass, with skeletal muscle being a key component. Although incretin-based therapies may improve the quality of skeletal muscle by reducing muscle fat infiltration, its function and strength remain underexplored. Exercise, adequate protein intake, and creatine supplementation mitigate these effects, whereas novel adjuncts such as myostatin/activin inhibitors and selective androgen receptor modulators show promise in early trials. Preserving muscle during incretin-based pharmacotherapy weight reduction is key to sustain long-term metabolic and cardiovascular benefits. Future trials should assess body composition, functional outcomes, and integrate muscle-preserving co-therapies into obesity management.

Indexed as

Anti-Obesity AgentsCardiovascular DiseasesIncretinsMuscle, SkeletalObesityBody CompositionHumansWeight LossAnti-Obesity AgentsIncretinsBody compositionCardiometabolic healthGLP-1 receptor agonistsLean mass preservationObesity pharmacotherapySkeletal muscle

Identifiers

What Socratic holds

Textmetadata
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.