Evidence mapPaperPMID 41409930Full record

ArticleCureus2025

Retinal Ganglion Cell Senescence Links Diabetes to Retinal Neurodegeneration.

Ayana Suzumura, Hideyuki Shimizu, Kazuhisa Yamada, Junya Ota, Seina Ito, Koji M Nishiguchi, Hiroki Kaneko

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In one paragraph

Article in Cureus, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

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0cells of the map it votes in
0citing papers in PubMed
field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

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Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

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0 citing papers in PubMed.

No citing paper in PubMed yet.

4 · The record

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PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

7 authors.

Ayana SuzumuraDepartment of Ophthalmology, Nagoya University Graduate School of Medicine, Nagoya, JPN.
Hideyuki ShimizuDepartment of Ophthalmology, Nagoya University Graduate School of Medicine, Nagoya, JPN.
Kazuhisa YamadaDepartment of Ophthalmology, Nagoya University Graduate School of Medicine, Nagoya, JPN.
Junya OtaDepartment of Ophthalmology, Nagoya University Graduate School of Medicine, Nagoya, JPN.
Seina ItoDepartment of Ophthalmology, Nagoya University Graduate School of Medicine, Nagoya, JPN.
Koji M NishiguchiDepartment of Ophthalmology, Nagoya University Graduate School of Medicine, Nagoya, JPN.
Hiroki KanekoDepartment of Ophthalmology, Hamamatsu University School of Meidicine, Hamamatsu, JPN.

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Background Diabetic retinopathy (DR) is a leading cause of blindness worldwide and traditionally considered a microvascular complication. However, accumulating evidence indicates that retinal neurodegeneration is also crucial in DR pathogenesis. Retinal ganglion cells (RGCs), the output neurons of the retina, are particularly vulnerable to diabetic stress. Cellular senescence has been implicated in diabetes-related tissue damage, but its contribution to RGC degeneration remains unclear. We hypothesized that diabetes contributes to retinal neurodegeneration by inducing senescence in RGCs. Methods In streptozotocin (STZ)-induced diabetic mice, retinal function was assessed via full-field electroretinography (ERG), and molecular changes were evaluated in senescence markers. The expression of p16

Indexed as

diabetic retinal neurodegenerationdiabetic retinopathyp16 generetinal ganglion cellsenescence-associated secretory phenotype (sasp)

Identifiers

PMID41409930
PMCPMC12706507

What Socratic holds

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.