Evidence mapPaperPMID 41416347Full record

ReviewBioMed research international2025

Targeting SIRT1: A Potential Strategy for Combating Severe COVID-19.

Hajar Shokri-Afra, Fatemeh Saber Jeyvan, Zeinab Barartabar, Parisa Khanicheragh, Elham Yousefi Abdolmaleki, Davod Ilbeigi, Hadis Musavi, Yalda Malekzadegan

Abstract readReview
In one paragraph

Review in BioMed research international, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 4 papers.

0numbers the graph read from it
0cells of the map it votes in
4citing papers in PubMed
field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

4 citing papers in PubMed.

  1. Review
  2. Review
  3. Article
  4. Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

8 authors.

Hajar Shokri-AfraGut and Liver Research Center, Non-Communicable Diseases Institute, Mazandaran University of Medical Sciences, Sari, Iran, mazums.ac.ir.ORCID https://orcid.org/0000-0001-9558-7104
Fatemeh Saber JeyvanDepartment of Biology, School of Sciences, University of Guilan, Rasht, Iran, guilan.ac.ir.ORCID https://orcid.org/0000-0002-8649-5686
Zeinab BarartabarCellular and Molecular Biology Research Center, Health Research Institute, Babol University of Medical Sciences, Babol, Iran, mubabol.ac.ir.ORCID https://orcid.org/0000-0003-0000-9198
Parisa KhanicheraghDepartment of Clinical Biochemistry and Laboratory Medicine, School of Medicine, Tabriz University of Medical Sciences, Tabriz, Iran, tbzmed.ac.ir.
Elham Yousefi AbdolmalekiGut and Liver Research Center, Non-Communicable Diseases Institute, Mazandaran University of Medical Sciences, Sari, Iran, mazums.ac.ir.ORCID https://orcid.org/0000-0002-6493-1512
Davod IlbeigiDepartment of Clinical Biochemistry, School of Medicine, Torbat Heydariyeh University of Medical Sciences, Torbat Heydariyeh, Iran, thums.ac.ir.ORCID https://orcid.org/0000-0002-4031-4181
Hadis MusaviDepartment of Clinical Biochemistry and Medical Genetics, Faculty of Medicine, Mazandaran University of Medical Sciences, Sari, Iran, mazums.ac.ir.ORCID https://orcid.org/0000-0003-1860-2387
Yalda MalekzadeganDepartment of Microbiology and Parasitology, School of Medicine, Bushehr University of Medical Sciences, Bushehr, Iran, bpums.ac.ir.ORCID https://orcid.org/0000-0002-4461-9900

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Sirtuin 1 (SIRT1) is a crucial regulator of cellular processes, including inflammation, metabolism, and stress responses, playing a significant role in the body's defense mechanisms. During SARS-CoV-2 infection, SIRT1 plays a crucial role in modulating the immune response. This protein helps to enhance the antiviral response through deacetylating key transcription factors and regulating proinflammatory cytokines, thereby reducing the cytokine storm (an overwhelming immune response) associated with severe COVID-19 cases. SIRT1 influences the expression of angiotensin-converting enzyme 2 (ACE2), the primary receptor for SARS-CoV-2, thereby potentially mitigating viral entry and replication. Natural activators of SIRT1, such as resveratrol, have been shown to enhance its activity, offering promising avenues for therapeutic interventions aimed at bolstering the immune response during COVID-19. Understanding the multifaceted role of SIRT1 in human defense mechanisms against SARS-CoV-2 could pave the way for innovative strategies to manage COVID-19 and similar viral infections, emphasizing the importance of SIRT1 as a potential target for future therapeutic approaches.

Indexed as

COVID-19COVID-19 Drug TreatmentSirtuin 1Angiotensin-Converting Enzyme 2Antiviral AgentsGene Expression RegulationHumansInflammationMolecular Targeted TherapyResveratrolVirus InternalizationAngiotensin-Converting Enzyme 2Antiviral AgentsResveratrolSIRT1 protein, humanSirtuin 1COVID-19cytokine storminflammationSARS-CoV-2signaling pathwayssirtuin 1

Identifiers

PMID41416347
PMCPMC12709655

What Socratic holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.