ReviewOncology reports2026
Research progress on the regulation of ferroptosis in NPC (Review).
Review in Oncology reports, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 1 paper.
What it found
Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.
The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
The trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.
Who cites it
1 citing paper in PubMed.
- Ferroptosis in colorectal cancer: Molecular mechanisms and regulatory crosstalk with therapeutic prospects (Review).Oncology reports · 2026Review
Corrections and comments
PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.
Authors and funding
5 authors.
Funding
No grant is acknowledged in the PubMed record.
Abstract
Ferroptosis is a novel form of iron‑dependent programmed apoptosis, characterized by dysregulated iron metabolism, impaired antioxidant defense systems and accumulation of lipid peroxidation products. Nasopharyngeal carcinoma (NPC) cells exhibit marked susceptibility to ferroptosis, and its induction can effectively suppress tumor progression, offering a potential therapeutic strategy for NPC. At the molecular level, ferroptosis‑related genes [such as Solute Carrier Family 7 Member 11 (SLC7A11), Glutamate‑Cysteine Ligase Modifier Subunit (GCLM) and Glutamate‑Cysteine Ligase Catalytic Subunit (GCLC)] are notably upregulated in NPC tissues compared with normal tissues, and their overexpression associates with poor patient prognosis, suggesting their utility as diagnostic or prognostic biomarkers. The present review systematically summarizes the molecular mechanisms of ferroptosis, elucidates its role in NPC pathogenesis and discusses ferroptosis‑targeted therapeutic approaches for NPC.
Indexed as
Identifiers
What Socratic holds
Registered trials
Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.