Evidence mapPaperPMID 41419000Full record

ArticleAmerican journal of physiology. Endocrinology and metabolism2026

Nr4a1 β-cell specific deletion impairs glucose tolerance in female mice.

Jacob A Herring, Adam G Wynn, Talon J Aitken, Nathan D Walker, Kate E Whalen, Jeffery S Tessem

Abstract read
In one paragraph

Article in American journal of physiology. Endocrinology and metabolism, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

0numbers the graph read from it
0cells of the map it votes in
0citing papers in PubMed
field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

0 citing papers in PubMed.

No citing paper in PubMed yet.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

6 authors.

Jacob A HerringDepartment of Microbiology and Molecular Biology, Brigham Young University, Provo, Utah, United States.ORCID 0000-0002-0084-1091
Adam G WynnDepartment of Nutrition, Dietetics and Food Science, Brigham Young University, Provo, Utah, United States.
Talon J AitkenDepartment of Nutrition, Dietetics and Food Science, Brigham Young University, Provo, Utah, United States.
Nathan D WalkerDepartment of Microbiology and Molecular Biology, Brigham Young University, Provo, Utah, United States.
Kate E WhalenDepartment of Microbiology and Molecular Biology, Brigham Young University, Provo, Utah, United States.
Jeffery S TessemDepartment of Microbiology and Molecular Biology, Brigham Young University, Provo, Utah, United States.ORCID 0000-0003-3081-3187

Funding

Nr4a1 and the expansion of functional beta-cell massR15DK144832 · BRIGHAM YOUNG UNIVERSITY · 2025 to 2025
$568k
HHS | National Institutes of Health (NIH) 1R15DK144832HHS | National Institutes of Health (NIH) R15DK124835NIDDK NIH HHS R15 DK124835NIDDK NIH HHS R15 DK144832
6 · The paper itself

Abstract

The Nr4a family of transcription factors controls the expression of genes essential for fuel metabolism and cellular proliferation. The loss of Nr4a1 in the 832/13 INS-1 β-cell line diminishes mitochondrial respiration, decreases ATP production, and impairs glucose-stimulated insulin secretion. Nr4a1 overexpression increases β-cell proliferation, and full-body Nr4a1 knockout mice have decreased β-cell area. Thus, we hypothesize that β-cell Nr4a1 expression plays a critical role in diabetes disease progression. Here we report the effects of β-cell specific

Indexed as

Glucose IntoleranceInsulin-Secreting CellsNuclear Receptor Subfamily 4, Group A, Member 1AnimalsDiet, High-FatEstrogensFemaleGlucose Tolerance TestInsulinInsulin SecretionMaleMiceMice, Inbred C57BLMice, KnockoutEstrogensInsulinNr4a1 protein, mouseNuclear Receptor Subfamily 4, Group A, Member 1estrogenhigh-fat dietinsulin secretionNr4a1β-cell

Identifiers

PMID41419000
PMCPMC12857568

What Socratic holds

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Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.