Evidence mapPaperPMID 41428987Full record

ArticleAnnals of neurology2026

Senescent Cell Clearance Ameliorates Temporal Lobe Epilepsy and Associated Spatial Memory Deficits in Mice.

Tahiyana Khan, David J McFall, Abbas I Hussain, Logan A Frayser, Timothy P Casilli, Meaghan C Steck, Irene Sanchez-Brualla, Noah M Kuehn, Michelle Cho, Jacqueline A Barnes and 3 more

Abstract read
In one paragraph

Article in Annals of neurology, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 2 papers.

0numbers the graph read from it
0cells of the map it votes in
2citing papers in PubMed
field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

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Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

2 citing papers in PubMed.

  1. Article
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4 · The record

Corrections and comments

5 · Who and what money

Authors and funding

13 authors.

Tahiyana Khan *Interdisciplinary Program in Neuroscience, Georgetown University, Washington, DC, USA.
David J McFall *Interdisciplinary Program in Neuroscience, Georgetown University, Washington, DC, USA.
Abbas I HussainDepartment of Pharmacology and Physiology, Georgetown University, Washington, DC, USA.
Logan A FrayserDepartment of Pharmacology and Physiology, Georgetown University, Washington, DC, USA.
Timothy P CasilliDepartment of Pharmacology and Physiology, Georgetown University, Washington, DC, USA.
Meaghan C SteckDepartment of Pharmacology and Physiology, Georgetown University, Washington, DC, USA.
Irene Sanchez-BruallaDepartment of Pharmacology and Physiology, Georgetown University, Washington, DC, USA.ORCID 0000-0002-1992-150X
Noah M KuehnDepartment of Pharmacology and Physiology, Georgetown University, Washington, DC, USA.
Michelle ChoDepartment of Pharmacology and Physiology, Georgetown University, Washington, DC, USA.
Jacqueline A BarnesDepartment of Neurology, Georgetown University, Washington, DC, USA.
Brent T HarrisInterdisciplinary Program in Neuroscience, Georgetown University, Washington, DC, USA.
Stefano ViciniInterdisciplinary Program in Neuroscience, Georgetown University, Washington, DC, USA.
Patrick A ForcelliInterdisciplinary Program in Neuroscience, Georgetown University, Washington, DC, USA.ORCID 0000-0003-1763-060X

Funding

Georgetown University Initiative for Maximizing Student Development (IMSD)T32GM144880 · NIGMS · GEORGETOWN UNIVERSITY · 2023 to 2025
$849k
Pharmacological Sciences Training Program (PSTP)T32GM142520 · GEORGETOWN UNIVERSITY · 2025 to 2025
$321k
Evaluating the contribution of senescent cells in a model of post-traumatic epilepsyF30NS143374 · GEORGETOWN UNIVERSITY · 2025 to 2025
$37k
NIGMS NIH HHS T32 GM142520NIGMS NIH HHS T32 GM144880NINDS NIH HHS F30 NS143374NINDS NIH HHS F99 NS129108NINDS NIH HHS R21 NS125552
6 · The paper itself

Abstract

objectiveThe pharmacological treatment of temporal lobe epilepsy (TLE), a disorder characterized by recurrent seizures and cognitive dysfunction, is limited to symptomatic control. Identifying novel targets to modify disease progression is of great clinical and translational interest. Cellular senescence has been recently implicated in the development and progression of other neurodegenerative diseases, but its role in TLE is unstudied.

methodsWe first investigated cellular senescence markers in resected hippocampi from patients with medically intractable TLE through multiplexed immunofluorescence. We next used a mouse model of TLE (pilocarpine induced status epilepticus [SE]) for a combination of immunohistochemistry, behavioral testing, and electroencephalogram (EEG) monitoring. We implemented 2 strategies for removal of senescent cells (SCs), a genetic mouse model allowing for targeted senolysis, and a pharmacological approach using dasatinib and quercetin.

resultsWe found a 5-fold elevation of senescent glia in human TLE cases as compared with controls. In mice, we found increases in senescence markers at both the transcript and protein level and predominantly expressed in microglia, which developed within 2 weeks following SE. Senolytic treatment produced a 50% reduction in SCs, rescued long-term potentiation deficits, normalized spatial memory impairments, reduced seizures, and protected a third of animals from epilepsy.

interpretationOur data demonstrate that SCs accumulate in both human TLE and in a mouse model of TLE and suggest that clearing SCs may be a viable strategy to reduce seizures and associated cognitive comorbidities. ANN NEUROL 2026;99:1059-1075.

Indexed as

Cellular SenescenceEpilepsy, Temporal LobeMemory DisordersSpatial MemoryAdultAnimalsDasatinibDisease Models, AnimalElectroencephalographyFemaleHippocampusHumansMaleMiceMice, Inbred C57BLMiddle AgedDasatinibPilocarpineQuercetinSenotherapeutics

Identifiers

PMID41428987
PMCPMC13198775

What Socratic holds

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Registered trials

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.