Evidence mapPaperPMID 41454404Full record

ArticleMalaria journal2025

Experimental evolution of Plasmodium yoelii in single and helminth-coinfected mice.

Aloïs Dusuel, Luc Bourbon, Emma Groetz, Mickaël Rialland, Benjamin Roche, Bruno Faivre, Gabriele Sorci

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Article in Malaria journal, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

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1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

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Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

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4 · The record

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5 · Who and what money

Authors and funding

7 authors.

Aloïs Dusuel *CTM - Center for Translational and Molecular Medicine, INSERM UMR 1231, Université Bourgogne Europe, 21000, Dijon, France.
Luc Bourbon *Biogéosciences, CNRS UMR 6282, Université Bourgogne Europe, 6 Boulevard Gabriel, 21000, Dijon, France.
Emma GroetzCTM - Center for Translational and Molecular Medicine, INSERM UMR 1231, Université Bourgogne Europe, 21000, Dijon, France.
Mickaël RiallandCTM - Center for Translational and Molecular Medicine, INSERM UMR 1231, Université Bourgogne Europe, 21000, Dijon, France.
Benjamin RocheMIVEGEC, IRD, CNRS, Université de Montpellier, 34090, Montpellier, France.
Bruno FaivreBiogéosciences, CNRS UMR 6282, Université Bourgogne Europe, 6 Boulevard Gabriel, 21000, Dijon, France.
Gabriele SorciBiogéosciences, CNRS UMR 6282, Université Bourgogne Europe, 6 Boulevard Gabriel, 21000, Dijon, France. gabriele.sorci@u-bourgogne.fr.

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

backgroundCoinfection has the potential to affect key traits describing the infection dynamics, the severity of the disease and in fine parasite fitness. However, despite its pervasiveness, experimental work investigating how parasites adapt to the conditions provided by a coinfected host is mostly missing.

methodsWe adopted an experimental evolution approach to investigate if coinfection with the nematode Heligmosomoides polygyrus (Hp) affected the infection dynamics and virulence of the murine malaria parasite Plasmodium yoelii (Py). To this purpose, lines of Py were passaged either in single infected hosts (SI-lines) or in hosts that had been previously infected with Hp (COI-lines). After five and seven passages, the infection dynamics and virulence of evolved lines were compared to the ancestral Py population during single infection trials. COI-lines were also used to infect hosts during coinfection trials, allowing us to compare within-host Py replication when the environment during the evaluation trials matched the environment experienced during the passages and when the two environments were mismatched.

resultsWe found that serial passages increased parasitemia and Py virulence, due to the competitive advantage of genotypes with the fastest replication rate, but SI-lines and COI-lines had relatively similar replication rate and virulence. Hosts infected with evolved lines of Py were also less tolerant (steeper slope between red blood cell counts and parasitemia) but there was no difference between SI-lines and COI-lines. Finally, we found that when COI-lines were used during single infection trials (mismatched environments), they had a slower early replication rate compared to matched-environment trials.

conclusionsWe did not find strong evidence supporting a divergence between the virulence of SI-lines and COI-lines, possibly due to the cost of virulence paid by COI-lines. However, Py rapidly adapted to the environmental conditions provided by single infected or coinfected hosts, as shown by the slower replication rate found in mismatched-environment trials.

Indexed as

Biological EvolutionCoinfectionMalariaParasitemiaPlasmodium yoeliiStrongylida InfectionsAnimalsDisease Models, AnimalFemaleHost-Parasite InteractionsMiceMice, Inbred C57BLNematospiroides dubiusSeverity of Illness IndexVirulenceCoinfectionHeligmosomoides polygyrusMismatched environmentsNematodesParasite adaptationPlasmodium yoeliiVirulence

Identifiers

PMID41454404
PMCPMC12849330

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.