Evidence map›Paper›PMID 41465730›Full record

ArticleLife (Basel, Switzerland)2025

The Neutrophil-to-Albumin Ratio (NAR) Reflects the Severity of the Post-CABG Inflammatory Response and Is Associated with a Pre-Existing Pro-Inflammatory Monocyte Profile.

Mikhail A Popov, Siarhei A Dabravolski, Vladislav V Dontsov, Sergei A Vzvarov, Evgeniy G Agafonov, Dmitriy I Zybin, Alexandra K Kharabet, Olga V Radchenkova, Dmitriy R Saveliev, Victoria P Pronina and 12 more

Abstract read
In one paragraph

Article in Life (Basel, Switzerland), 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 3 papers.

0numbers the graph read from it
0cells of the map it votes in
3citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

3 citing papers in PubMed.

  1. Article
  2. Article
  3. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

22 authors.

Mikhail A PopovM.F. Vladimirsky Moscow Regional Clinical Research Institute, Schepkina Street 61/2, Moscow 129110, Russia.ORCID 0000-0002-0316-8410
Siarhei A DabravolskiDepartment of Biotechnology Engineering, Braude Academic College of Engineering, Snunit 51, P.O. Box 78, Karmiel 2161002, Israel.ORCID 0000-0002-0547-6310
Vladislav V DontsovM.F. Vladimirsky Moscow Regional Clinical Research Institute, Schepkina Street 61/2, Moscow 129110, Russia.ORCID 0000-0003-2904-5213
Sergei A VzvarovM.F. Vladimirsky Moscow Regional Clinical Research Institute, Schepkina Street 61/2, Moscow 129110, Russia.ORCID 0009-0007-2548-7029
Evgeniy G AgafonovM.F. Vladimirsky Moscow Regional Clinical Research Institute, Schepkina Street 61/2, Moscow 129110, Russia.
Dmitriy I ZybinM.F. Vladimirsky Moscow Regional Clinical Research Institute, Schepkina Street 61/2, Moscow 129110, Russia.
Alexandra K KharabetM.F. Vladimirsky Moscow Regional Clinical Research Institute, Schepkina Street 61/2, Moscow 129110, Russia.
Olga V RadchenkovaM.F. Vladimirsky Moscow Regional Clinical Research Institute, Schepkina Street 61/2, Moscow 129110, Russia.ORCID 0009-0004-4372-3302
Dmitriy R SavelievM.F. Vladimirsky Moscow Regional Clinical Research Institute, Schepkina Street 61/2, Moscow 129110, Russia.ORCID 0009-0003-1571-4583
Victoria P ProninaM.F. Vladimirsky Moscow Regional Clinical Research Institute, Schepkina Street 61/2, Moscow 129110, Russia.ORCID 0000-0002-2348-1500
Svetlana S VerkhovaInstitute of General Pathology and Pathophysiology, Baltiyskaya Street 8, Moscow 125315, Russia.ORCID 0000-0002-7953-0586
Nikita G NikiforovInstitute of General Pathology and Pathophysiology, Baltiyskaya Street 8, Moscow 125315, Russia.ORCID 0000-0002-2082-2429
Yegor S ChegodaevInstitute of General Pathology and Pathophysiology, Baltiyskaya Street 8, Moscow 125315, Russia.ORCID 0009-0005-7710-2202
Alexander D ZhuravlevInstitute of General Pathology and Pathophysiology, Baltiyskaya Street 8, Moscow 125315, Russia.
Daiana B ErdyneevaInstitute of General Pathology and Pathophysiology, Baltiyskaya Street 8, Moscow 125315, Russia.
Yegor E YegorovInstitute of General Pathology and Pathophysiology, Baltiyskaya Street 8, Moscow 125315, Russia.
Elena E SigalevaInstitute of General Pathology and Pathophysiology, Baltiyskaya Street 8, Moscow 125315, Russia.
Milena I KolotevaInstitute of General Pathology and Pathophysiology, Baltiyskaya Street 8, Moscow 125315, Russia.ORCID 0000-0002-7499-9473
Ekaterina V SilinaI. M. Sechenov First Moscow State Medical University (Sechenov University), Moscow 119991, Russia.ORCID 0000-0002-0246-5149
Victor A StupinPirogov Russian National Research Medical University, Moscow 117997, Russia.ORCID 0000-0002-9522-8061
Alexander V IvanovInstitute of General Pathology and Pathophysiology, Baltiyskaya Street 8, Moscow 125315, Russia.ORCID 0000-0002-2424-6115
Dmitriy V ShumakovM.F. Vladimirsky Moscow Regional Clinical Research Institute, Schepkina Street 61/2, Moscow 129110, Russia.ORCID 0000-0003-4204-8865

Funding

Ministry of Science and Higher Education of the Russian Federation FGFU-2025-0006
6 · The paper itself

Abstract

backgroundThe systemic inflammatory response to coronary artery bypass grafting (CABG) is highly variable and a key driver of complications. We hypothesised that a pre-existing pro-inflammatory immune state, characterised by a skewed monocyte profile, 'primes' patients for an exaggerated response. This pilot prospective study aimed to test this hypothesis and to evaluate the Neutrophil-to-Albumin Ratio (NAR) as an integrated biomarker of this response, comparing it against the established Neutrophil-to-Lymphocyte Ratio (NLR).

methodsIn this pilot prospective, single-centre pilot study, we enrolled 34 patients with multivessel coronary artery disease (CAD) scheduled for off-pump CABG and 20 control subjects. Preoperatively, peripheral blood monocyte subsets were quantified by flow cytometry. Neutrophil, lymphocyte, and albumin levels were measured before and after surgery to calculate NAR and NLR. Multivariable linear regression was used to test for independent predictors of the inflammatory response.

resultsPreoperatively, CAD patients exhibited a reduced percentage of the classical monocyte subpopulation (

conclusionA patient's preoperative immune profile, specifically the degree of monocyte skew, is an independent predictor of the acute inflammatory response to CABG. This finding supports a 'priming' mechanism in high-risk patients. While NAR and NLR perform similarly as monitoring tools, the independent link between the underlying immunology and the postoperative outcome suggests that combining preoperative immunophenotyping with simple biomarker monitoring could offer a powerful new strategy for personalised risk stratification in cardiac surgery.

Indexed as

biomarkercardiac surgerycoronary artery bypass graftingcoronary artery diseaseinflammationmonocyte subsetsneutrophil-to-albumin ratioperioperative care

Identifiers

PMID41465730
PMCPMC12733900

What Socratic holds

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Registered trials

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.