Evidence map›Paper›PMID 41469877›Full record

ArticleLipids in health and disease2025

Lecithin coenzyme Q10 restores mitochondrial dynamics and alleviates hepatic dysfunction in high-fat Diet-Fed db/db mice.

Chen-Ling Kuo, Chih-Chung Wu, Yu-Shan Cheng, Ching-Shan Huang, Chin-San Liu, Shih-Li Su

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Article in Lipids in health and disease, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 1 paper.

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1citing papers in PubMed
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1 · What the graph read from it

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3 · Its place in the literature

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1 citing paper in PubMed.

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4 · The record

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5 · Who and what money

Authors and funding

6 authors.

Chen-Ling KuoVascular Medicine and Diabetes Research Center, Institute of ATP, Changhua Christian Hospital, Changhua, Taiwan.
Chih-Chung WuDepartment of Food and Nutrition, Providence University, Taichung, Taiwan.
Yu-Shan ChengVascular Medicine and Diabetes Research Center, Institute of ATP, Changhua Christian Hospital, Changhua, Taiwan.
Ching-Shan HuangCenter of Regenerative Medicine and Tissue Repair, Institute of ATP, Changhua, Taiwan.
Chin-San LiuDepartment of Neurology, Institute of ATP, Changhua, Taiwan.
Shih-Li SuVascular Medicine and Diabetes Research Center, Institute of ATP, Changhua Christian Hospital, Changhua, Taiwan. 89933@cch.org.tw.

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

BACKGROUND/

objectivesThis study investigated the metabolic and pathological effects of a high-fat diet (HFD) in db/db mice and evaluated the therapeutic efficacy of various Coenzyme Q10 (CoQ10) products. We aimed to determine whether HFD-induced mitochondrial damage can be improved by different CoQ10 products through either repairing mitochondrial injury or increasing mitochondrial bioenergy, thereby addressing the root cause of oxidative stress. METHODS AND

resultsPlasma biochemical analyses revealed that HFD induced hyperglycemia, elevated hepatic transaminases [aspartate aminotransferase (AST), alanine aminotransferase (ALT)], and dyslipidemia. Lecithin coenzyme Q10 (SoQ10) significantly improved these parameters, especially in reducing AST (255 ± 73.8 U/L vs. 138 ± 29.4 U/L, p < 0.05), ALT (87.8 ± 17.3 U/L vs. 79.2 ± 11.9 U/L, p < 0.05), and triglyceride levels (142.0 ± 37.0 mg/dL vs. 15.5 ± 2.5 mg/dL, p < 0.05), demonstrating greater efficacy than standard CoQ10. Histological evaluation showed that HFD caused marked hepatic steatosis and inflammatory infiltration. Oil Red O staining further confirmed excessive lipid deposition in the livers of HFD-fed mice. Both Q10 treatments decreased lipid droplet accumulation (p < 0.05), with SoQ10 showing a greater reduction (p < 0.05), indicating its potential to alleviate hepatic steatosis. Further assessments indicated that gene expression analyses showed that HFD upregulated lipid metabolism-related genes [lipoprotein lipase (LPL), peroxisome proliferator-activated receptor-γ (PPAR-γ), sterol regulatory element-binding protein-1 (SREBP-1), alkaline ceramidase 2 (ACER2)] (p < 0.05), indicating an imbalance between lipogenesis and lipolysis. SoQ10 modulated these genes and further enhanced ceramide synthase 2 (CERS2) expression, suggesting a role in reestablishing hepatic lipid homeostasis. Additionally, SoQ10 significantly upregulated genes associated with mitochondrial biogenesis peroxisome proliferator-activated receptor-γ coactivator-1α (PGC-1α), mitochondrial transcription factor A (TFAM)] (p < 0.05) and mitochondrial dynamics [mitofusin-2 (MFN2), optic atrophy type 1 long isoform (OPA1-L)] as well as fission [dynamin-related protein 1 (DRP1), mitochondrial fission protein 1 (Fis1)] (p < 0.05), indicating a potential to restore mitochondrial structural balance. In contrast, conventional CoQ10 had a more limited effect, particularly on fusion-related gene expression.

conclusionsSoQ10 demonstrated superior therapeutic potential over conventional CoQ10 in ameliorating hepatic metabolic dysfunction, oxidative mitochondrial damage, and disturbances in lipid metabolism and mitochondrial dynamics induced by a high-fat diet.

Indexed as

LecithinsLiverMitochondrial DynamicsUbiquinoneAlanine TransaminaseAnimalsAspartate AminotransferasesDiet, High-FatDyslipidemiasMaleMiceMice, Inbred C57BLOxidative StressPeroxisome Proliferator-Activated Receptor Gamma Coactivator 1-alphaPPAR gammaSterol Regulatory Element Binding Protein 1Alanine TransaminaseAspartate Aminotransferasescoenzyme Q10LecithinsPeroxisome Proliferator-Activated Receptor Gamma Coactivator 1-alphaPPAR gammaSterol Regulatory Element Binding Protein 1TriglyceridesUbiquinoneCoenzyme Q10Diabetes mellitusLipid metabolismMitochondrial dysfunction

Identifiers

PMID41469877
PMCPMC12874728

What Socratic holds

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LicenceCC BY-NC-ND
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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.