Evidence mapPaperPMID 41480113Full record

ReviewFrontiers in microbiology2025

Gut-muscle axis crosstalk in age-related sarcopenia: mechanisms and therapeutic targets.

Ling-Li Gao, Yan Chen, Ting Dai, Jie Zheng, Shuo-Shuo Su, Yi-Xun Chen, Li-Dian Chen, Jing Gao, Xiao-Dong Feng

Abstract readReview
In one paragraph

Review in Frontiers in microbiology, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 6 papers.

0numbers the graph read from it
0cells of the map it votes in
6citing papers in PubMed
field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

6 citing papers in PubMed.

  1. Kimchi ProbioticJournal of microbiology and biotechnology · 2026
    Article
  2. Review
  3. Review
  4. Review
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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

9 authors.

Ling-Li GaoDepartment of Rehabilitation, The First Affiliated Hospital of Henan University of Chinese Medicine, Zhengzhou, China.
Yan ChenDepartment of Rehabilitation, The First Affiliated Hospital of Henan University of Chinese Medicine, Zhengzhou, China.
Ting DaiDepartment of Rehabilitation, The First Affiliated Hospital of Henan University of Chinese Medicine, Zhengzhou, China.
Jie ZhengSchool of Rehabilitation Medicine, Henan University of Chinese Medicine, Zhengzhou, China.
Shuo-Shuo SuSchool of Rehabilitation Medicine, Henan University of Chinese Medicine, Zhengzhou, China.
Yi-Xun ChenSchool of Rehabilitation Medicine, Henan University of Chinese Medicine, Zhengzhou, China.
Li-Dian ChenSchool of Rehabilitation Medicine, Henan University of Chinese Medicine, Zhengzhou, China.
Jing GaoDepartment of Rehabilitation, The First Affiliated Hospital of Henan University of Chinese Medicine, Zhengzhou, China.
Xiao-Dong FengDepartment of Rehabilitation, The First Affiliated Hospital of Henan University of Chinese Medicine, Zhengzhou, China.

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

The interplay between gut microbiota and sarcopenia has emerged as a cutting-edge research topic in the medical field, garnering significant attention. Sarcopenia is an age-related syndrome characterized by a progressive decline in skeletal muscle mass, strength, and function, which profoundly impacts the quality of life in older adults and imposes substantial socioeconomic burdens on many counties. Accumulating evidence indicates that alterations in the gut microbiota are not only linked to various intestinal disorders but also to aging-associated conditions, such as sarcopenia. The gut microbiota plays a pivotal role in regulating skeletal muscle homeostasis via its metabolic products and is increasingly recognized as a potential pathophysiological factor contributing to sarcopenia development. Skeletal muscle, functioning as both a motor and endocrine organ, secretes myokines that exert critical regulatory effects on the gut microbiota. In sarcopenic individuals, reduced secretion of myokines correlates with decreased microbial diversity and compositional shifts, marked by diminished beneficial microbes and increased potentially harmful species. This establishes a vicious cycle of gut dysbiosis-sarcopenia-gut dysbiosis. Modulation of the gut microbiota has been demonstrated to enhance muscle mass and function in elderly patients with sarcopenia. Metabolites derived from the gut microbiota, such as amino acids, lipopolysaccharides, and short-chain fatty acids, are known to modulate skeletal muscle protein metabolism by influencing anabolic and catabolic pathways. Nevertheless, the bidirectional mechanisms underlying the relationship between gut microbiota and age-related sarcopenia remain incompletely understood. In this review, we aim to: (1) integrate current knowledge regarding the bidirectional interaction between sarcopenia and gut microbiota; (2) summarize existing management strategies for age-related sarcopenia based on this interaction.

Indexed as

age-related sarcopeniacrosstalkgut-muscle axisinflammationmicrobial metabolitesmyokinesneuroendocrine systemtherapeutic targets

Identifiers

PMID41480113
PMCPMC12755243

What Socratic holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.