In one paragraphArticle in Pflugers Archiv : European journal of physiology, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.
0numbers the graph read from it
0cells of the map it votes in
0citing papers in PubMed
–field-weighted citation impact
What it found
Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.
The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
2 · The registryThe trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.
3 · Its place in the literatureWho cites it
0 citing papers in PubMed.
No citing paper in PubMed yet.
4 · The recordCorrections and comments
5 · Who and what moneyAuthors and funding
11 authors.
Daniel EssigkeDepartment of Internal Medicine, Division of Diabetology, Endocrinology and Nephrology, University Hospital Tübingen, Otfried-Mueller-Str.10, Tübingen, 72076, Germany.ORCID 0000-0002-1473-377X M Zaher KaloDepartment of Internal Medicine, Division of Diabetology, Endocrinology and Nephrology, University Hospital Tübingen, Otfried-Mueller-Str.10, Tübingen, 72076, Germany.
Lingsi KongDepartment of Internal Medicine, Division of Diabetology, Endocrinology and Nephrology, University Hospital Tübingen, Otfried-Mueller-Str.10, Tübingen, 72076, Germany.
Matthias WörnDepartment of Internal Medicine, Division of Diabetology, Endocrinology and Nephrology, University Hospital Tübingen, Otfried-Mueller-Str.10, Tübingen, 72076, Germany.
Mohammad-Khaled SaadDepartment of Internal Medicine, Division of Diabetology, Endocrinology and Nephrology, University Hospital Tübingen, Otfried-Mueller-Str.10, Tübingen, 72076, Germany.
Kingsley OmageDepartment of Internal Medicine, Division of Diabetology, Endocrinology and Nephrology, University Hospital Tübingen, Otfried-Mueller-Str.10, Tübingen, 72076, Germany.ORCID 0000-0001-7450-6731 Bernhard N BohnertDepartment of Internal Medicine, Division of Diabetology, Endocrinology and Nephrology, University Hospital Tübingen, Otfried-Mueller-Str.10, Tübingen, 72076, Germany.ORCID 0000-0003-0169-3948 Andreas L BirkenfeldDepartment of Internal Medicine, Division of Diabetology, Endocrinology and Nephrology, University Hospital Tübingen, Otfried-Mueller-Str.10, Tübingen, 72076, Germany.ORCID 0000-0003-1407-9023 John P AtkinsonDepartment of Medicine, Division of Rheumatology, Washington University School of Medicine, St. Louis, MO, USA.ORCID 0000-0002-2514-3441 Xiaobo WuDepartment of Medicine, Division of Rheumatology, Washington University School of Medicine, St. Louis, MO, USA.ORCID 0000-0002-6703-2272 Ferruh ArtuncDepartment of Internal Medicine, Division of Diabetology, Endocrinology and Nephrology, University Hospital Tübingen, Otfried-Mueller-Str.10, Tübingen, 72076, Germany. ferruh.artunc@med.uni-tuebingen.de.ORCID 0000-0002-3777-9316 Funding
NIH HHS NIH R35-GM136352
6 · The paper itselfAbstract
The complement component C3, factor B (FB) and factor D (FD) belong to the alternative complement pathway and have been identified in urine samples from nephrotic mice. However, it is not yet known whether these factors are involved in mediating sodium retention in nephrotic syndrome (NS). Here we used a genetic mouse model of NS based on an inducible podocin deletion (Nphs2
Indexed as
Complement Pathway, AlternativeNephrotic SyndromeSodiumAnimalsComplement C3Complement Factor BDisease Models, AnimalEpithelial Sodium ChannelsIntracellular Signaling Peptides and ProteinsMaleMembrane ProteinsMiceMice, Inbred C57BLMice, KnockoutComplement C3Complement Factor BEpithelial Sodium ChannelsIntracellular Signaling Peptides and ProteinsMembrane ProteinsNPHS2 proteinSodiumAlternative complement pathwayEdemaEpithelial sodium channelNephrotic syndromeSodium retention
Identifiers
PMID41483032
PMCPMC12764515
What Socratic holds
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