Evidence mapPaperPMID 41501437Full record

ReviewAnnals of biomedical engineering2026

Precision Nanotechnology: Revolutionizing Therapeutic Strategies Against Drug-Resistant Breast Cancer.

ZahraSadat Razavi, Arefeh Mottaghi, Lyudmila Dmitrieva, Shiva Fallahianshafiei, Erfan Olfat, Nahid Ahmadi

Abstract readReview
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In one paragraph

Review in Annals of biomedical engineering, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 2 papers.

0numbers the graph read from it
0cells of the map it votes in
2citing papers in PubMed
field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

2 citing papers in PubMed.

  1. Article
  2. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

6 authors.

ZahraSadat RazaviPhysiology Research Center, Iran University Medical Sciences, Tehran, Iran. Farnaz.razavi@iau.ir.ORCID http://orcid.org/0000-0003-2404-3227
Arefeh Mottaghi *Student Research Committee, Iran University of Medical Sciences, Tehran, Iran.
Lyudmila DmitrievaDepartment of Infectious Diseases, Institute of Public Health named after F.F. Erisman, Sechenov First Moscow State Medical University (Sechenov University), Moscow, Russia.
Shiva Fallahianshafiei *Department of Pharmacy, Ayatollah Amoli Branch, Islamic Azad University, Amol, Iran.
Erfan OlfatDepartment of Materials Science and Engineering, K. N. Toosi University of Technology, Tehran, Iran.
Nahid AhmadiDepartment of Immunology, School of Medicine,Lorestan University of Medical Sciences, Kamalvand Campus, Khorramabad, Iran.

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Drug resistance, which arises from a variety of biological mechanisms such as drug efflux by ATP-binding cassette transporters, epithelial-to-mesenchymal transition (EMT), the presence of cancer stem cells (CSCs), immune system evasion, and changes in the tumor microenvironment (TME), poses serious treatment challenges for breast cancer. This paper offers a thorough analysis of various resistance mechanisms, going into great detail into the molecular, cellular, and genetic contributors. We next look at how nanotechnology-based approaches, such multifunctional nanocarriers, smart nanoparticle systems, and targeted drug delivery, can be developed to get around these resistance mechanisms, building on this biological basis. The use of particular nanotechnology platforms (such as liposomes, micelles, and dendrimers) to target CSCs, circumvent efflux pumps, alter immunological responses, and improve treatment precision is highlighted. Important issues in clinical translation and regulatory considerations are also covered in the study. This two-part investigation seeks to improve therapeutic outcomes for drug-resistant breast cancer by bridging the gap between molecular insight and nanotechnological innovation.

Indexed as

Antineoplastic AgentsBreast NeoplasmsDrug Delivery SystemsDrug Resistance, NeoplasmNanomedicineAnimalsFemaleHumansNanoparticlesNanotechnologyNeoplastic Stem CellsTumor MicroenvironmentAntineoplastic AgentsBreast cancerControlled drug releaseDrug resistanceMultidrug resistance (MDR)NanoparticlesTargeted drug delivery

Identifiers

What Socratic holds

Textmetadata
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.