Evidence map›Paper›PMID 41501697›Full record

ArticleBMC infectious diseases2026

Distinct systemic cytokine signatures define symptomatic malaria in children: insights from a longitudinal study in Togo.

Fagdéba David Bara, Hombamane Christelle Simfele, Tédalomba Koudema, Solinba Bako, Mona-Liza E Sakyi, Albert Yao Kudakpo, Charles Mensah, Esther Donkor, Frederica P Malm, Manuel Ritter and 3 more

Abstract read
In one paragraph

Article in BMC infectious diseases, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 1 paper.

0numbers the graph read from it
0cells of the map it votes in
1citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

1 citing paper in PubMed.

  1. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

13 authors.

Fagdéba David BaraWest African Centre for Cell Biology of Infectious Pathogens, University of Ghana, Legon-Accra, Ghana.
Hombamane Christelle SimfeleUnité de Recherche en Immunologie et Immunomodulation (UR2IM)/Laboratoire de Microbiologie et de Contrôle de Qualité des Denrées Alimentaires/Ecole Supérieure des Techniques Biologiques et alimentaires (ESTBA), Université de Lomé, BP 1515, Lomé, Togo.
Tédalomba KoudemaUnité de Recherche en Immunologie et Immunomodulation (UR2IM)/Laboratoire de Microbiologie et de Contrôle de Qualité des Denrées Alimentaires/Ecole Supérieure des Techniques Biologiques et alimentaires (ESTBA), Université de Lomé, BP 1515, Lomé, Togo.
Solinba BakoUnité de Recherche en Immunologie et Immunomodulation (UR2IM)/Laboratoire de Microbiologie et de Contrôle de Qualité des Denrées Alimentaires/Ecole Supérieure des Techniques Biologiques et alimentaires (ESTBA), Université de Lomé, BP 1515, Lomé, Togo.
Mona-Liza E SakyiWest African Centre for Cell Biology of Infectious Pathogens, University of Ghana, Legon-Accra, Ghana.
Albert Yao KudakpoWest African Centre for Cell Biology of Infectious Pathogens, University of Ghana, Legon-Accra, Ghana.
Charles MensahWest African Centre for Cell Biology of Infectious Pathogens, University of Ghana, Legon-Accra, Ghana.
Esther DonkorWest African Centre for Cell Biology of Infectious Pathogens, University of Ghana, Legon-Accra, Ghana.
Frederica P MalmWest African Centre for Cell Biology of Infectious Pathogens, University of Ghana, Legon-Accra, Ghana.
Manuel RitterInstitute for Medical Microbiology, Immunology and Parasitology (IMMIP), University Hospital Bonn (UKB), Bonn, Germany.
Gordon A AwandareWest African Centre for Cell Biology of Infectious Pathogens, University of Ghana, Legon-Accra, Ghana.
Gnatoulma Katawa *Unité de Recherche en Immunologie et Immunomodulation (UR2IM)/Laboratoire de Microbiologie et de Contrôle de Qualité des Denrées Alimentaires/Ecole Supérieure des Techniques Biologiques et alimentaires (ESTBA), Université de Lomé, BP 1515, Lomé, Togo. mahkatawa@yahoo.fr.ORCID http://orcid.org/0000-0002-4131-5274
Lucas N Amenga-Etego *West African Centre for Cell Biology of Infectious Pathogens, University of Ghana, Legon-Accra, Ghana. lamengaetego@ug.edu.gh.ORCID http://orcid.org/0000-0003-4468-0506

Funding

Bill & Melinda Gates Foundation INV-050873WACCBIP-World Bank African Centres of Excellence (ACE) WACCBIP+NCDs: Awandare
6 · The paper itself

Abstract

backgroundMalaria remains a significant public health challenge in sub-Saharan Africa (SSA), particularly among children, where immune responses and clinical manifestations to Plasmodium falciparum infection vary widely. Understanding the interplay between parasitemia, immune responses, and clinical outcomes is essential to improving case management. This study aimed to identify systemic cytokine signatures that define symptomatic malaria in children, by analyzing cytokine dynamics over time and their association with parasite burden and clinical status in children aged 6–10 years in Togo.

methodsA longitudinal cohort study was conducted in the Prefecture d’Agoè, Togo, from November 2023 to February 2024. Sixty children were enrolled and treated with artemether-lumefantrine and albendazole. Weekly monitoring was performed over 12 weeks using microscopy and qPCR for P. falciparum detection. Systemic cytokines, including IL-1β, IL-6, IFN-γ, IL-17 A, IL-5, and IL-10, were measured by Sandwich ELISA. Associations between cytokine concentrations, parasite densities, and cycle threshold (Ct) values were assessed using Spearman’s correlation.

resultsOf the 60 children, 25% (n = 15) tested positive for P. falciparum by qPCR, while 15% (n = 9) had microscopy-confirmed infections. Overall, 33.3% (n = 5) progressed to symptomatic malaria; the remainder were asymptomatic carriers. Symptomatic cases exhibited significantly elevated IL-6 (vs. non-infected, p = 0.0004; vs. asymptomatic, p = 0.0003; vs. recovered, p = 0.0037), IFN-γ (vs. non-infected, p < 0.0001; vs. asymptomatic, p = 0.0048), and IL-10 levels (vs. non-infected, p < 0.0001; vs. recovered, p = 0.0004). IL-10 levels were also higher in asymptomatic children compared to non-infected (p = 0.0013), while IFN-γ was elevated in recovered cases (vs. non-infected, p = 0.0309). In symptomatic children, IL-10 levels showed a strong negative correlation with Ct values (r = -0.973, p = 0.001) and a strong positive correlation with parasite density (r = 0.911, p = 0.001). IFN-γ exhibited moderate correlations with both, Ct values (r = -0.442, p = 0.037) and parasite density (r = 0.518, p = 0.027).

conclusionThis study identifies distinct cytokine signatures, particularly elevated IL-10 and IFN-γ, that characterize symptomatic malaria in children and correlate with parasite burden. These cytokines may serve as prognostic biomarkers and offer insights for targeted interventions in paediatric malaria.

Indexed as

CytokinesMalaria, FalciparumAlbendazoleAntimalarialsArtemether, Lumefantrine Drug CombinationChildFemaleHumansInterferon-gammaInterleukin-10Longitudinal StudiesMaleParasitemiaPlasmodium falciparumTogoAlbendazoleAntimalarialsArtemether, Lumefantrine Drug CombinationCytokinesInterferon-gammaInterleukin-10CytokinesIFN-γIL-10MalariaPaediatric immune responseParasite burdenPlasmodium falciparum

Identifiers

PMID41501697
PMCPMC12870030

What Socratic holds

Textmetadata
LicenceCC BY-NC-ND
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Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.