Evidence map›Paper›PMID 41510106›Full record

ArticleTranslational cancer research2025

Machine learning screening for disulfidptosis genes-associated immunosuppression status in osteosarcoma and rhabdomyosarcoma.

Lin Yu, Fanli Lin

Abstract read
In one paragraph

Article in Translational cancer research, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

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0cells of the map it votes in
0citing papers in PubMed
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1 · What the graph read from it

What it found

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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

0 citing papers in PubMed.

No citing paper in PubMed yet.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

2 authors.

Lin YuDepartment of Pediatric Surgery, Children's Medical Center, The Affiliated Hospital of Southwest Medical University, Luzhou, China.ORCID https://orcid.org/0000-0001-7666-635X
Fanli LinDepartment of Hematology, The Affiliated Hospital of Southwest Medical University, Luzhou, China.ORCID https://orcid.org/0009-0006-3492-6161

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Background: The recent identification of disulfidptosis, a novel form of cell death, offers significant potential for advancing cancer therapeutics. Osteosarcoma (OS) and rhabdomyosarcoma (RMS) are prevalent malignant sarcomas in children and young adults, yet their molecular underpinnings remain poorly understood, hampering treatment options. This study aimed to delineate the role of disulfidptosis in these malignancies and to identify key molecular determinants of prognosis. Methods: We analyzed extensive RNA transcriptome data from the Gene Expression Omnibus (GEO) and the Therapeutically Applicable Research to Generate Effective Treatments (TARGET) databases. Non-negative matrix factorization (NMF) clustering was employed to identify disulfidptosis-associated molecular subtypes. Furthermore, we integrated 100 machine-learning algorithms to pinpoint core prognostic genes. Results: Based on disulfidptosis-related genes, we identified a distinct immunosuppressive subtype in both OS and RMS, characterized by significantly poorer immune cell infiltration. A robust prognostic signature comprising five genes ( Conclusions: Our findings illuminate the landscape of disulfidptosis in OS and RMS, revealing a novel immunosuppressive subtype and a defined five-gene signature for risk stratification. The identification of

Indexed as

disulfidptosisimmunosuppressionmachine learningOsteosarcoma (OS)rhabdomyosarcoma (RMS)

Identifiers

PMID41510106
PMCPMC12776226

What Socratic holds

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Registered trials

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.