Evidence map›Paper›PMID 41521073›Full record

ReviewBMB reports2026

Epigenetic aging and rejuvenation of the brain: drivers, consequences, and interventions.

Seunghyun Lee, SeungA Cho, Seung-Kyoon Kim, Jae-Hyun Yang

Abstract readReview
In one paragraph

Review in BMB reports, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

0numbers the graph read from it
0cells of the map it votes in
0citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

0 citing papers in PubMed.

No citing paper in PubMed yet.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

4 authors.

Seunghyun LeeDepartment of Brain & Cognitive Sciences, Korea Advanced Institute of Science and Technology (KAIST), Daejeon 34141, Korea.
SeungA ChoDepartment of Brain & Cognitive Sciences, Korea Advanced Institute of Science and Technology (KAIST), Daejeon 34141; Department of Microbiology and Molecular Biology, Chungnam National University, Daejeon 34134, Korea.
Seung-Kyoon KimDepartment of Convergent Bioscience and Informatics, and Graduate School of Biological Sciences, Chungnam National University, Daejeon 34134, Korea.
Jae-Hyun YangDepartment of Brain & Cognitive Sciences, Korea Advanced Institute of Science and Technology (KAIST), Daejeon 34141, Korea.

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Evolution has tuned epigenetic resilience to preserve chromatin organization, transcriptional networks, and cellular identity under relentless stress. Over time, however, all eukaryotic life faces an inevitable rise in entropy that erodes the chromatin landscape at the genomic scale. This entropic decay of epigenetic information, epigenetic aging, is a primary driver of biological aging and systemic dysfunction. The brain is particularly vulnerable to epigenetic aging, with post-mitotic neurons accumulating lifelong chromatin erosion, and the glial epigenome drifting toward pro-inflammatory states. Defining the drivers and consequences of epigenetic aging in the brain forms the basis for restoring youthful chromatin landscapes, cellular identity, and cognitive capacity. [BMB Reports 2026; 59(1): 27-39].

Indexed as

AgingBrainEpigenesis, GeneticRejuvenationAnimalsChromatinHumansChromatin

Identifiers

PMID41521073
PMCPMC12867171

What Socratic holds

Textmetadata
LicenceCC BY-NC
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.