ArticleJournal of the American Heart Association2026
Heart Failure Risk in Patients With Systemic Autoimmune Inflammatory Diseases.
Article in Journal of the American Heart Association, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 1 paper.
What it found
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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
The trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.
Who cites it
1 citing paper in PubMed.
- Heart Disease in Older Women: Unique Challenges in Diagnosis and Management.Current cardiology reports · 2026Review
Corrections and comments
PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.
Authors and funding
3 authors.
Funding
No grant is acknowledged in the PubMed record.
Abstract
backgroundCardiac disease, including heart failure (HF), is a significant cause of morbidity and mortality for patients with systemic autoimmune inflammatory diseases (SAIDs). We sought to describe HF risk among patients with SAIDs and to investigate associations between cardiovascular drug use and incident HF.
methodsWe conducted a retrospective cohort study of electronic health records of 182 795 adult patients with SAIDs including systemic sclerosis, systemic lupus erythematosus, rheumatoid arthritis, psoriatic arthritis, inflammatory bowel disease, and HIV from 2000 to 2020 and track their risks of new-onset incident HF in a large multidisciplinary health care institution.
resultsSystemic sclerosis (adjusted hazard ratio [aHR], 2.81 [95% CI, 2.57-3.08];
conclusionsPatients with systemic sclerosis, systemic lupus erythematosus, and rheumatoid arthritis had increased risk of incident HF independent of traditional risk factors, indicating an underlying autoimmune mechanism of cardiac involvement. Beta-blocker use was associated with decreased incident HF, indicating a potentially cardioprotective effect in SAIDs.
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