ArticleBMC genomics2026
Comprehensive transcriptomic profiling identifies key regulatory genes mediating phytohormone signaling pathways during seed germination in Chenopodium quinoa.
Article in BMC genomics, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.
What it found
Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.
The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
The trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.
Who cites it
0 citing papers in PubMed.
No citing paper in PubMed yet.
Corrections and comments
PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.
Authors and funding
9 authors.
Funding
Abstract
backgroundPre-harvest sprouting (PHS) significantly reduces the yield and quality of Chenopodium quinoa (quinoa). A key determinant of PHS resistance is the balance between seed dormancy and germination, a process primarily regulated by phytohormones.
resultsTo elucidate the molecular mechanisms underlying hormone-mediated germination regulation, we performed transcriptome sequencing on Jingli 1 quinoa seeds 6 h after treatment with six phytohormones: abscisic acid (ABA), indole-3-acetic acid (IAA), jasmonic acid (JA), gibberellic acid (GA₃), brassinolide (BR), and 6-benzylaminopurine (6-BA). The results showed that ABA, IAA, and JA significantly inhibited germination, with a maximum inhibition rate of 36.73%. In contrast, optimal concentrations of GA₃ (45 µM), BR (20 µM), and 6-BA (4.44 µM) promoted germination, with a maximum promotion rate of 22.67%. Transcriptome analysis identified 4,738 differentially expressed genes (DEGs), which were significantly enriched in pathways such as plant hormone signal transduction, starch and sucrose metabolism, and terpenoid backbone biosynthesis. Furthermore, we identified 102 coregulated DEGs, revealing intricate hormone signaling networks (such as ABA-GA-JA and JA-IAA-6-BA). Importantly, we pinpointed 20 core regulatory genes (including CqWRKY33, CqAnxD3, CqbHLH18L, CqMYB-V, CqSES, CqHMGCS, CqMVK1/2, CqCKX7-1/-2, CqWAT1-1/-2, CqORR9/10, CqBNM2AL, CqSAUR72L, CqRAV1L, CqABCG31L, Cq2-ODD, and CqBG7Sg2) that showed antagonistic expression patterns in response to promotive versus inhibitory hormones.
conclusionsThis study systematically elucidates the multi-hormone regulatory network underlying quinoa seed germination, thereby enhancing our understanding of phytohormone-mediated regulatory mechanisms in quinoa. It also identifies promising candidate genes for breeding pre-harvest sprouting (PHS)-resistant quinoa varieties.
Indexed as
Identifiers
What Socratic holds
Registered trials
Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.