Evidence mapPaperPMID 41574187Full record

ReviewFrontiers in physiology2025

Cold exposure and the cardiovascular system: from physiological adaptation to pathological risk.

Yao Li, Jiawei Wu, Yinli Xu, Junbo Dong, Bo Xing, Yiwen Wang, Zijun Zhou, Boxuan Sun, Jiahui Li, Liming Yu and 1 more

Abstract readReview
In one paragraph

Review in Frontiers in physiology, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 8 papers.

0numbers the graph read from it
0cells of the map it votes in
8citing papers in PubMed
field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

8 citing papers in PubMed.

  1. Article
  2. Review
  3. Article
  4. Article
  5. Article
  6. Review
  7. Combined effects of apparent low temperature and PMFrontiers in public health · 2026
    Article
  8. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

11 authors.

Yao LiCollege of Medicine and Biological Information Engineering, Northeastern University, Shenyang, China.
Jiawei WuState Key Laboratory of Frigid Zone Cardiovascular Disease, Department of Cardiovascular Surgery, General Hospital of Northern Theater Command, Shenyang, China.
Yinli XuState Key Laboratory of Frigid Zone Cardiovascular Disease, Department of Cardiovascular Surgery, General Hospital of Northern Theater Command, Shenyang, China.
Junbo DongState Key Laboratory of Frigid Zone Cardiovascular Disease, Department of Cardiovascular Surgery, General Hospital of Northern Theater Command, Shenyang, China.
Bo XingState Key Laboratory of Frigid Zone Cardiovascular Disease, Department of Cardiovascular Surgery, General Hospital of Northern Theater Command, Shenyang, China.
Yiwen WangCollege of Medicine and Biological Information Engineering, Northeastern University, Shenyang, China.
Zijun ZhouState Key Laboratory of Frigid Zone Cardiovascular Disease, Department of Cardiovascular Surgery, General Hospital of Northern Theater Command, Shenyang, China.
Boxuan SunState Key Laboratory of Frigid Zone Cardiovascular Disease, Department of Cardiovascular Surgery, General Hospital of Northern Theater Command, Shenyang, China.
Jiahui LiState Key Laboratory of Frigid Zone Cardiovascular Disease, Department of Cardiovascular Surgery, General Hospital of Northern Theater Command, Shenyang, China.
Liming YuState Key Laboratory of Frigid Zone Cardiovascular Disease, Department of Cardiovascular Surgery, General Hospital of Northern Theater Command, Shenyang, China.
Huishan WangCollege of Medicine and Biological Information Engineering, Northeastern University, Shenyang, China.

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Cold stress is the therapeutic paradox of cardiovascular medicine: both an established environmental trigger of acute death and a physiological stimulus for powerful adaptation. In this review, we address this paradox critically. The response to cold challenge, which ranges from sympathetic activation and hemodynamic stress to adaptive brown adipose tissue recruitment and cardiac metabolic remodeling, which can be pathological or protective. We synthesize evidence from specific cardiovascular diseases, such as coronary disease and heart failure, and summarize molecular pathways of metabolic, inflammatory, and electrophysiological effects. Native adaptive mechanisms and their therapeutic potential as templates are also discussed. Through synthesis of these multi-faceted avenues, this article builds upon a theoretical basis to propose a mechanistic model for the seasonal regulation of CVD and to outline emerging, cold-based research avenues.

Indexed as

autonomic nervous systembrown adipose tissuecardiovascular diseasescold exposuremetabolic remodelingmolecular mechanisms

Identifiers

PMID41574187
PMCPMC12819719

What Socratic holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.