Evidence mapPaperPMID 41574758Full record

ArticleJournal of diabetes investigation2026

Increased C-reactive protein is associated with corneal nerve loss and painful neuropathic symptoms in Type 2 diabetes mellitus.

Georgios Ponirakis, Ibrahim Al-Janahi, Einas Elgassim, Ioannis N Petropoulos, Hoda Gad, Adnan Khan, Hadeel B Zaghloul, Hamda Ali, Mashhood A Siddique, Sidra Abdulshakoor and 18 more

Abstract read
In one paragraph

Article in Journal of diabetes investigation, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

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0citing papers in PubMed
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1 · What the graph read from it

What it found

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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

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Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

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0 citing papers in PubMed.

No citing paper in PubMed yet.

4 · The record

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5 · Who and what money

Authors and funding

28 authors.

Georgios PonirakisDepartment of Medicine, Weill Cornell Medicine-Qatar, Qatar Foundation, Doha, Qatar.ORCID https://orcid.org/0000-0002-6936-1248
Ibrahim Al-JanahiNational Diabetes Center, Hamad General Hospital, Hamad Medical Corporation, Doha, Qatar.
Einas ElgassimDepartment of Medicine, Weill Cornell Medicine-Qatar, Qatar Foundation, Doha, Qatar.
Ioannis N PetropoulosDepartment of Medicine, Weill Cornell Medicine-Qatar, Qatar Foundation, Doha, Qatar.
Hoda GadDepartment of Medicine, Weill Cornell Medicine-Qatar, Qatar Foundation, Doha, Qatar.
Adnan KhanDepartment of Medicine, Weill Cornell Medicine-Qatar, Qatar Foundation, Doha, Qatar.
Hadeel B ZaghloulDepartment of Medicine, Weill Cornell Medicine-Qatar, Qatar Foundation, Doha, Qatar.
Hamda AliNational Diabetes Center, Hamad General Hospital, Hamad Medical Corporation, Doha, Qatar.
Mashhood A SiddiqueNational Diabetes Center, Hamad General Hospital, Hamad Medical Corporation, Doha, Qatar.
Sidra AbdulshakoorQatar Biobank, Qatar Foundation, Hamad Medical City, Doha, Qatar.
Jenneth M ConcepcionQatar Biobank, Qatar Foundation, Hamad Medical City, Doha, Qatar.
Sara H KhalfallaQatar Biobank, Qatar Foundation, Hamad Medical City, Doha, Qatar.
Iynas S A ElaminQatar Biobank, Qatar Foundation, Hamad Medical City, Doha, Qatar.
Abeer T H AlZawqariQatar Biobank, Qatar Foundation, Hamad Medical City, Doha, Qatar.
Tania BibiQatar Biobank, Qatar Foundation, Hamad Medical City, Doha, Qatar.
Marwa A El DeebQatar Biobank, Qatar Foundation, Hamad Medical City, Doha, Qatar.
Lina H M AhmedDepartment of Medicine, Weill Cornell Medicine-Qatar, Qatar Foundation, Doha, Qatar.
Youssra DakrouryDepartment of Medicine, Weill Cornell Medicine-Qatar, Qatar Foundation, Doha, Qatar.
Abeer M M El ShewehyDepartment of Medicine, Weill Cornell Medicine-Qatar, Qatar Foundation, Doha, Qatar.
Iuliia SalivonNational Diabetes Center, Hamad General Hospital, Hamad Medical Corporation, Doha, Qatar.
Mohammad Qasim MinhasDepartment of Medicine, Weill Cornell Medicine-Qatar, Qatar Foundation, Doha, Qatar.ORCID https://orcid.org/0009-0006-8722-5051
Yara IsmailDepartment of Medicine, Weill Cornell Medicine-Qatar, Qatar Foundation, Doha, Qatar.
Mahmoud A ZirieNational Diabetes Center, Hamad General Hospital, Hamad Medical Corporation, Doha, Qatar.
Yousuf Al-AnsariNational Diabetes Center, Hamad General Hospital, Hamad Medical Corporation, Doha, Qatar.
Mariam A O Al-MalaheemNational Diabetes Center, Hamad General Hospital, Hamad Medical Corporation, Doha, Qatar.
Dorothy J QuadrosNational Diabetes Center, Hamad General Hospital, Hamad Medical Corporation, Doha, Qatar.
Stephen L AtkinRoyal College of Surgeons in Ireland Bahrain, Adliya, Kingdom of Bahrain.
Rayaz A MalikDepartment of Medicine, Weill Cornell Medicine-Qatar, Qatar Foundation, Doha, Qatar.ORCID https://orcid.org/0000-0002-7188-8903

Funding

Qatar National Research Fund BMRP-5726113101Qatar National Research Fund NPRP 8-315-3-065
6 · The paper itself

Abstract

AIMS/

introductionTo investigate the association between C-reactive protein (CRP) with sensory symptoms and deficits and measures of small fiber damage. MATERIALS AND

methodsAdults with and without T2D underwent corneal confocal microscopy (CCM) and assessment of vibration perception threshold (VPT), DN4 symptom questionnaire, and fasting blood tests for CRP and metabolic markers.

resultsOf the 122 participants, 77 had T2D of whom 23 (29.9%) had DPN, and 45 were controls. CRP levels were significantly higher in those with T2D without and with DPN compared to controls (5.6 ± 2.9 and 5.4 ± 4.0 vs 3.8 ± 2.9 mg/L, P = 0.008 and P = 0.046, respectively), with no difference between those with and without DPN (P = 0.894). Higher CRP was independently associated with lower corneal nerve fiber density (CNFD) (β = -5.5 fibers/mm

conclusionsElevated CRP is associated with small nerve fiber loss and positive neuropathic symptoms in T2D. These findings suggest that CRP may help identify individuals with inflammation-driven DPN who could benefit from targeted interventions.

Indexed as

CorneaC-Reactive ProteinDiabetes Mellitus, Type 2Diabetic NeuropathiesNeuralgiaAgedBiomarkersCase-Control StudiesFemaleHumansMaleMicroscopy, ConfocalMiddle AgedNerve FibersPrognosisBiomarkersC-Reactive ProteinCorneal confocal microscopyC‐reactive proteinDiabetic peripheral neuropathy

Identifiers

PMID41574758
PMCPMC12950922

What Socratic holds

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.