Evidence mapPaperPMID 41575476Full record

ArticleJournal of physiology and biochemistry2026

Procyanidin B2 and an autochthonous apple pulp extract modulate oxidative stress and PPARγ expression on an in vitro model of lipid steatosis in HepG2 cells.

Adrián Millán-Laleona, Marta Lopez-Yus, Silvia Lorente-Cebrián, Jose M Arbones-Mainar, Carlota Gómez-Rincón, Víctor López

Abstract read
In one paragraph

Article in Journal of physiology and biochemistry, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

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1 · What the graph read from it

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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

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3 · Its place in the literature

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4 · The record

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5 · Who and what money

Authors and funding

6 authors.

Adrián Millán-LaleonaDepartment of Pharmacy, Faculty of Health Sciences, Universidad San Jorge, Zaragoza, 50830, Spain.
Marta Lopez-YusInstituto de Investigación Sanitaria (IIS) Aragon, Zaragoza, 50009, Spain.
Silvia Lorente-CebriánInstituto de Investigación Sanitaria (IIS) Aragon, Zaragoza, 50009, Spain.
Jose M Arbones-MainarInstituto de Investigación Sanitaria (IIS) Aragon, Zaragoza, 50009, Spain.
Carlota Gómez-RincónDepartment of Pharmacy, Faculty of Health Sciences, Universidad San Jorge, Zaragoza, 50830, Spain.
Víctor LópezDepartment of Pharmacy, Faculty of Health Sciences, Universidad San Jorge, Zaragoza, 50830, Spain. ilopez@usj.es.

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Metabolic dysfunction-associated steatotic liver disease (MASLD) is characterized by hepatic steatosis alongside metabolic comorbidities like type 2 diabetes mellitus (T2DM), dyslipidaemia, overweight, or obesity. Liver damage could be prevented promoting consumption of fruits and vegetables, the main source of phenolic compounds. These molecules have been previously related with antioxidant and anti-obesity properties. Therefore, the objective of this study is to determine the antioxidant capacity and potential preventive activity of an autochthonous apple pulp extract, Amarilla de Octubre, and its main phenolic compound, procyanidin B2 (PB2), in an in vitro model of liver steatosis. Antioxidant activity was assessed by xanthine/xanthine oxidase (X/XO) system and nitric oxide scavenging production. Monoamine oxidase A (MAO-A) inhibition was also determined due to its implication in detoxification processes in liver. Human hepatocytes (HepG2 cell line) were selected to test their potential cytotoxicity, oxidative stress situations (ROS production) and the potential effect in lipid metabolism by overloading cells with oleic acid. Fat accumulation and gene expression related to lipid metabolism (PPARγ, CD36 and FAS) were also analysed by Oil Red O and qPCR respectively. PB2 showed promising results as an antioxidant compound in scavenging and enzymatic inhibition-related experiments. However, Amarilla de Octubre not only reduce fat accumulation, but also modulates the expression of PPARγ, CD36, and FAS and demonstrates slightly lower efficacy than PB2 in scavenging and enzymatic inhibition-related experiments. These results highlight the potential of apples to mitigate pathogenic conditions associated with metabolic disorders such as obesity, emphasizing the role of dietary polyphenols in this protective effect.

Indexed as

AntioxidantsBiflavonoidsCatechinFatty LiverMalusOxidative StressPlant ExtractsPPAR gammaProanthocyanidinsFruitHepatocytesHep G2 CellsHumansLipid MetabolismAntioxidantsBiflavonoidsCatechinPlant ExtractsPPAR gammaProanthocyanidinsprocyanidin B2AppleHepG2Lipid metabolismMASLDPhenolic compoundsProcyanidin B2

Identifiers

PMID41575476
PMCPMC12830479

What Socratic holds

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.