ReviewFrontiers in cell and developmental biology2025
Immune imbalance and autoimmune mechanisms in plasma cell mastitis: current perspectives.
Review in Frontiers in cell and developmental biology, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 3 papers.
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Who cites it
3 citing papers in PubMed.
- Retinal and choroidal optical coherence tomography findings in gestational diabetes mellitus: a systematic review and meta-analysis.Graefe's archive for clinical and experimental ophthalmology = Albrecht von Graefes Archiv fur klinische und experimentelle Ophthalmologie · 2026Review
- Prolactin-Linked Plasma Cell-Macrophage Immune Phenotype in Synchronous Bilateral Plasma Cell Mastitis: A Two-Center Prediction Study with Tissue Correspondence.International journal of women's health · 2026Article
- Recent Advances in Pathogenesis, Diagnosis and Integrated Traditional Chinese Medicine Therapy for Plasma Cell Mastitis.International journal of general medicine · 2026Review
Corrections and comments
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Authors and funding
9 authors.
Funding
No grant is acknowledged in the PubMed record.
Abstract
Plasma cell mastitis (PCM), also termed ductal dilatation or periductal mastitis (PDM), is a benign, non-bacterial inflammatory breast disorder predominantly affecting non-lactating women. Its incidence continues to rise, yet diagnosis and therapeutic approaches remain under development. Consequently, elucidating the pathogenesis is essential for identifying the disease's root cause and facilitating breakthroughs. PCM is increasingly regarded as an autoimmune disorder, highlighting the crucial role of immune factors in its development; however, the precise immune mechanisms involved remain incompletely understood. Against these backgrounds, this review aims to: (1) discuss the contributions of various innate and adaptive immune cells within the PCM-associated immune microenvironment; (2) describe the relationship between imbalances in key immune cells (including M1/M2 macrophages, Th1/Th2 lymphocytes, Th17/Treg cells) and the progression of PCM; (3) explore potential future research areas, the underlying immunopathogenesis of PCM, and prospective therapeutic targets, the activation of the complement system, regulation of immune homeostasis, the role of immune cell circadian rhythms, and modulation of key immune pathways.
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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.