Evidence mapPaperPMID 41635458Full record

ArticleJournal of medicine and life2025

Modulation of metabolic syndrome components by oral semaglutide in hypothyroid-T2DM patients: a retrospective analysis.

Dana-Mihaela Tilici, Ruxandra-Mihaela Costinescu, Diana-Loreta Paun, Sorin Constantin Paun, Cristian Guja

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Article in Journal of medicine and life, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

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1 · What the graph read from it

What it found

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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

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3 · Its place in the literature

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4 · The record

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5 · Who and what money

Authors and funding

5 authors.

Dana-Mihaela TiliciDoctoral School, Carol Davila University of Medicine and Pharmacy, Bucharest, Romania.
Ruxandra-Mihaela CostinescuUniversity Emergency Hospital of Bucharest, Bucharest, Romania.
Diana-Loreta PaunCarol Davila University of Medicine and Pharmacy, Bucharest, Romania.
Sorin Constantin PaunBucharest Emergency Clinical Hospital, Bucharest, Romania.
Cristian GujaCarol Davila University of Medicine and Pharmacy, Bucharest, Romania.

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Metabolic syndrome (MetS) represents the concurrent manifestation of multiple cardiometabolic risk factors, including visceral obesity, hyperglycemia, hypertension, hypertriglyceridemia, and low HDL-cholesterol, cumulatively predisposing to accelerated atherosclerosis and type 2 diabetes mellitus (T2DM). Hypothyroidism frequently coexists with T2DM and further exacerbates insulin resistance (IR), lipid abnormalities, and systemic inflammation, increasing the prevalence and severity of MetS in this population. Oral semaglutide is a glucagon-like peptide-1 receptor agonist approved for T2DM management; however, its impact on MetS parameters in patients with coexisting hypothyroidism remains insufficiently explored. This study aimed to evaluate the effects of oral semaglutide on key MetS components in this high-risk subgroup. We conducted a single-center retrospective cohort study involving 51 adult patients with confirmed hypothyroidism and T2DM, on oral semaglutide (final dose = 14 mg daily) and monitored for 6 months. Clinical and biochemical parameters were analyzed, including glycated hemoglobin (HbA1c), body mass index (BMI), blood pressure, and lipid profile. At 6 months, mean HbA1c decreased by 6.7% (

Indexed as

Diabetes Mellitus, Type 2Glucagon-Like PeptidesHypoglycemic AgentsHypothyroidismMetabolic SyndromeAdministration, OralAdultAgedFemaleGlycated HemoglobinHumansMaleMiddle AgedRetrospective StudiesSemaglutideGlucagon-Like PeptidesGlycated HemoglobinHypoglycemic AgentsSemaglutideBMI, body mass indexdyslipidemiaGLP-1, glucagon-like peptide-1GLP-1 receptor agonistHbA1c, glycated hemoglobinHDL, high-density lipoproteinhypothyroidisminsulin resistanceIR, insulin resistanceLDL, low-density lipoproteinmetabolic syndromeMetS, metabolic syndromeoral semaglutideSAH, systemic arterial hypertensionSCH, subclinical hypothyroidismT2DM, type 2 diabetes mellitusTSH, thyroid-stimulating hormonetype 2 diabetes mellitus

Identifiers

PMID41635458
PMCPMC12863099

What Socratic holds

Texttitle and abstract
LicenceCC BY
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Registered trials

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.