Evidence map›Paper›PMID 41638428›Full record

ReviewThe Journal of biological chemistry2026

AHCY: A metabolic gatekeeper at the interface of methylation, redox balance, and cellular stress response.

Sarah C Stanhope, Vikki M Weake

Abstract readReview
In one paragraph

Review in The Journal of biological chemistry, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 2 papers.

0numbers the graph read from it
0cells of the map it votes in
2citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

2 citing papers in PubMed.

  1. Biomolecules · 2026
    Review
  2. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

2 authors.

Sarah C StanhopeDepartment of Biochemistry, Purdue University, West Lafayette, Indiana, USA.
Vikki M WeakeDepartment of Biochemistry, Purdue University, West Lafayette, Indiana, USA; Purdue Institute for Cancer Research, Purdue University, West Lafayette, Indiana, USA. Electronic address: vweake@purdue.edu.

Funding

Chromatin connects metabolism to circadian gene regulation in the aging eyeR01EY033734 · NEI · PURDUE UNIVERSITY · PI Vikki Marie Weake · 2023 to 2026
$1.5M
Characterizing Ahcy as a redox regulated enzyme in the Drosophila eye under oxidative stressF31EY036754 · NEI · PURDUE UNIVERSITY · PI GREGOR, SARAH · 2024 to 2025
$99k
NEI NIH HHS F31 EY036754NEI NIH HHS R01 EY033734
6 · The paper itself

Abstract

S-Adenosylhomocysteinase (AHCY, also known as SAHH) is a highly conserved enzyme that catalyzes the reversible hydrolysis of SAH into adenosine and homocysteine. As the sole enzyme capable of catalyzing this reaction, AHCY modulates cellular methylation potential required for DNA, RNA, and protein methyltransferase activity. Recent discoveries, however, expand its role well beyond this canonical function, positioning AHCY as a metabolic gatekeeper that integrates one-carbon metabolism with epigenetic regulation, RNA processing, nucleotide balance, and redox signaling. This review brings together mechanistic, structural, and regulatory insights into AHCY while critically evaluating diverse biochemical and biophysical methods for assaying its activity. Comparative structural analyses uncover conserved tetrameric organization alongside species-specific adaptations in oligomeric state, NAD

Indexed as

AdenosylhomocysteinaseStress, PhysiologicalAnimalsHumansMethylationOxidation-ReductionAdenosylhomocysteinaseAHCY protein, humanenzymatic regulationmethylation potentialone-carbon metabolismredox homeostasisS-adenosylhomocysteinase (AHCY)S-adenosylhomocysteinase hydrolase (SAHH)

Identifiers

PMID41638428
PMCPMC12955632

What Socratic holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.