Evidence map›Paper›PMID 41648862›Full record

ArticleFrontiers in bioinformatics2025

Cross-disease transcriptomic meta-analysis and network pharmacology reveal key therapeutic targets in rheumatoid arthritis, systemic lupus erythematosus and multiple sclerosis.

K Lakshmi, Sundararajan Vino

Abstract read
In one paragraph

Article in Frontiers in bioinformatics, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

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0citing papers in PubMed
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1 · What the graph read from it

What it found

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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

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3 · Its place in the literature

Who cites it

0 citing papers in PubMed.

No citing paper in PubMed yet.

4 · The record

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5 · Who and what money

Authors and funding

2 authors.

K LakshmiIntegrative Multiomics Lab, Department of Bio Sciences, School of Bio Sciences and Technology, Vellore Institute of Technology, Vellore, Tamil Nadu, India.
Sundararajan VinoIntegrative Multiomics Lab, Department of Bio Sciences, School of Bio Sciences and Technology, Vellore Institute of Technology, Vellore, Tamil Nadu, India.

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Autoimmune disease has a complex etiology that remains not fully understood. We aimed to identify highly perturbed DEGs and hub genes associated with autoimmune disease Rheumatoid Arthritis (RA), Systemic Lupus Erythematosus (SLE) and Multiple Sclerosis (MS). To find potentially lead to more effective therapies that target the root causes of these diseases. Materials and methods: Datasets for autoimmune diseases (RA, SLE, and MS) were collected from the GEO database. Differentially expressed genes were identified and subjected to meta-analysis to obtain common DEGs, which were then used for functional enrichment analysis GO and pathway analysis. A PPI network was constructed, and topology-based ranking identified hub genes. These hub genes were further analyzed through regulatory network analysis (TF and miRNA), gene-disease association studies, and drug-gene interaction analysis. Finally, molecular docking and molecular dynamics (MD) simulations were performed on the hub genes. Results: A total of 341 differentially expressed genes were identified, with 172 upregulated and 169 downregulated genes. Among these, eight hub genes Conclusion: These findings explored identifying the DEGs among shade of the autoimmune disease RA, SLE, MS, and this hub gene are associated with transcription factors are most crucial role play in the disease potentially clinical therapeutic targets of the autoimmune disease.

Indexed as

autoimmune diseasesdifferentially expressed genesfunctional enrichment analysismolecular dynamicstranscriptome

Identifiers

PMID41648862
PMCPMC12868135

What Socratic holds

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.