Evidence map›Paper›PMID 41656402›Full record

ArticlePharmacological reports : PR2026

Naringenin and quercetin alter mouse rod phototransduction kinetics in a manner consistent with phosphodiesterase-6 inhibition.

Lorenzo Cangiano, Claudia Gargini, Sabrina Asteriti

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Article in Pharmacological reports : PR, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

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5 · Who and what money

Authors and funding

3 authors.

Lorenzo CangianoDepartment of Translational Research, University of Pisa, Pisa, Italy.ORCID http://orcid.org/0000-0001-7145-1288
Claudia GarginiDepartment of Pharmacy, University of Pisa, Via Bonanno 6, 56126, Pisa, Italy.ORCID http://orcid.org/0000-0002-4992-9086
Sabrina AsteritiDepartment of Pharmacy, University of Pisa, Via Bonanno 6, 56126, Pisa, Italy. sabrina.asteriti@unipi.it.ORCID http://orcid.org/0000-0001-5713-0015

Funding

European Union Next Generation EU B83C22003930001
6 · The paper itself

Abstract

backgroundThe flavonoids naringenin and quercetin are promising therapeutic agents for a wide variety of diseases, including inherited retinal degenerations (IRDs), thanks to their antioxidant, anti-inflammatory, and other properties. Among these are their inhibition of some phosphodiesterase (PDE) isoforms. However, it is unknown how they affect the photoreceptor-specific PDE6, which is critical for phototransduction. This interaction is key for understanding their therapeutic potential in IRDs and the risk of visual side effects more broadly. We aimed to determine if they inhibit PDE6 in living mouse retinae at pharmacologically relevant concentrations.

methodsWe used ex vivo electroretinography to record pharmacologically isolated rod a-waves from mouse retinae at 37 °C. One retina received naringenin or quercetin, while the other received the vehicle. We analysed flash response parameters: light sensitivity, time-to-peak, amplification constant (derived by fitting responses with a biophysical model), and saturating amplitude.

resultsBoth flavonoids rapidly decreased amplification, in line with inhibition of light-activated PDE6 with a [Formula: see text] of 7.1 µM for naringenin and 15.1 µM for quercetin. Naringenin increased saturating amplitude, consistent with inhibiting dark-state PDE6. Quercetin induced a secondary slowing of phototransduction cascade termination kinetics independent of its effect on amplification, suggesting inhibition of a cascade shutdown protein.

conclusionsThese findings are key to evaluating the visual safety of high-dose flavonoid therapies. They also raise the possibility that direct effects on phototransduction could play a role in their neuroprotection in IRDs.

Indexed as

Cyclic Nucleotide Phosphodiesterases, Type 6FlavanonesPhosphodiesterase InhibitorsQuercetinRetinal Rod Photoreceptor CellsAnimalsElectroretinographyKineticsMiceMice, Inbred C57BLRetinaCyclic Nucleotide Phosphodiesterases, Type 6FlavanonesnaringeninPhosphodiesterase InhibitorsQuercetinFlavonoidsNaringeninPhosphodiesterase-6PhotoreceptorsQuercetin

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.