ArticleFEBS open bio2026
Identification of functional murine mitochondrial formyl peptides and their effects on myeloid-derived suppressor cell generation.
Article in FEBS open bio, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.
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Abstract
N-formyl peptides are cleavage products of bacterial and mitochondrial proteins, which effects on neutrophil activation have been extensively studied. Mitochondrial formyl peptides are also known to play a role in tumor immunity, but the molecular mechanism underlying this role remains largely unexplored. Our previous work suggested that tumor-derived mitochondrial formyl peptides promote tumor growth by enhancing myeloid-derived suppressor cell (MDSC)-mediated cytotoxic T-lymphocyte suppression. Therefore, we hypothesized that tumor-derived mitochondrial formyl peptides act directly on bone marrow cells to promote MDSC generation. In this study, we tested this hypothesis by first identifying five functional murine mitochondrial formyl peptides that induced intracellular Ca
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