ArticleActa neuropathologica communications2026
MGMT promoter methylation modulates the effect of residual tumor on survival after re-resection of recurrent glioblastoma.
Article in Acta neuropathologica communications, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.
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Abstract
Surgical re-resection for recurrent IDH–wildtype glioblastoma remains controversial, and no molecular biomarkers currently inform on tailoring the extent of cytoreduction and hence onco-functional balance at recurrence. On the other hand, while the prognostic relevance of O6-methylguanine-DNA-methyltransferase (MGMT) promoter methylation is established in glioblastoma. Its role in surgical decision-making at recurrence remains unclear. We analyzed a cohort of 153 patients who underwent re-resection for IDH-wildtype recurrent glioblastoma (WHO classification 2021) between 2015 and 2024. Volumetric analysis of contrast-enhancing (CE) tumor on pre- and early postoperative MRI was performed. Patients were stratified by MGMT promoter methylation status and residual tumor volume (RTV): 0 ml, < 1 ml, > 1 ml. Functional outcomes up to one year after surgery were longitudinally assessed using the National Institutes of Health Stroke Scale (NIHSS). There was no significant difference in RTV between MGMT-methylated (n = 58, 38%) and unmethylated patients (p > 0.999). The benefit from complete or near-complete resection was more pronounced in MGMT-unmethylated tumors, with a median postoperative survival of 288 days for 0 to 1 ml RTV versus 190 days > 1 ml (p = 0.024; difference = 98 days), compared to MGMT-methylated tumors with a median survival of 411 days versus 378 days (p = 0.043; difference = 33 days). MGMT-unmethylated patients with 0 ml RTV showed significantly higher KPS values six weeks postoperatively (p < 0.05) compared to higher RTVs. Postoperative deficits were comparable across groups. These findings identify MGMT promoter methylation as a clinically actionable molecular modifier of the oncological benefit of cytoreduction and support MGMT-stratified, precision neurosurgical strategies for recurrent glioblastoma, especially around functionally eloquent tumors.
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