Evidence map›Paper›PMID 41696109›Full record

ReviewWorld journal of diabetes2026

Metabolic dysfunction-associated steatotic liver disease and type 2 diabetes: Pathophysiology, diagnosis, and emerging therapeutic strategies.

Mithil Gowda Suresh, Safia Mohamed, Harinivaas S Geetha, Sushmita Prabhu, Nitin Trivedi, Zhu C Ng, Priyal D Mehta, Ajit S Brar, Aalam Sohal, Manjeet K Goyal and 2 more

Abstract readReview
In one paragraph

Review in World journal of diabetes, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 14 papers.

0numbers the graph read from it
0cells of the map it votes in
14citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

14 citing papers in PubMed.

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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

12 authors.

Mithil Gowda SureshDepartment of Internal Medicine, Saint Vincent Hospital, Worcester, MA 01608, United States.
Safia MohamedDepartment of Medicine and Surgery, University of Massachusetts Chan Medical School-Baystate Medical Center, Springfield, MA 01199, United States.
Harinivaas S GeethaDepartment of Internal Medicine, Saint Vincent Hospital, Worcester, MA 01608, United States.
Sushmita PrabhuDepartment of Internal Medicine, Saint Vincent Hospital, Worcester, MA 01608, United States.
Nitin TrivediDepartment of Internal Medicine, Saint Vincent Hospital, Worcester, MA 01608, United States.
Zhu C NgDepartment of Internal Medicine, Saint Vincent Hospital, Worcester, MA 01608, United States.
Priyal D MehtaDepartment of Internal Medicine, Saint Vincent Hospital, Worcester, MA 01608, United States.
Ajit S BrarDepartment of Internal Medicine, Michigan State University at Hurley Medical Center, Flint, MI 48503, United States.
Aalam SohalDepartment of Gastroenterology and Hepatology, Creighton University School of Medicine, Phoenix, AZ 85012, United States.
Manjeet K GoyalDepartment of Internal Medicine, Cleveland Clinic Akron General Hospital, Akron, OH 110029, United States.
Juniali HatwalDepartment of Internal Medicine, Post Graduate Institute of Medical Education and Research, Chandigarh 160012, India.
Akash BattaDepartment of Cardiology, Dayanand Medical College and Hospital, Ludhiana 141001, Punjab, India. akashbatta02@gmail.com.

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Metabolic dysfunction-associated steatotic liver disease (MASLD), the updated terminology for fatty liver disease linked to metabolic dysfunction, is highly prevalent among individuals with type 2 diabetes mellitus (T2DM). MASLD affects a majority of patients with T2DM and markedly increases the risk of fibrosis, cirrhosis, hepatocellular carcinoma, and cardiovascular mortality. The pathogenesis in diabetic populations reflects a convergence of insulin resistance, dyslipidemia, mitochondrial dysfunction, chronic inflammation, and genetic predisposition. Advances in non-invasive diagnostics, including elastography and serum biomarkers, enable earlier identification and staging of disease, though limitations remain in diabetic cohorts. Lifestyle modification is the cornerstone of therapy, yet emerging pharmacotherapies are reshaping the therapeutic landscape. Antidiabetic agents such as glucagon-like peptide-1 receptor agonists, sodium-glucose cotransporter-2 inhibitors, and pioglitazone show hepatic benefits beyond glycemic control, while novel agents and combination regimens are under active evaluation. This narrative review synthesizes current evidence on epidemiology, mechanisms, diagnostics, and therapeutics of MASLD in T2DM, and highlights future directions in precision medicine. Integration of multidisciplinary care is essential to address this converging epidemic.

Indexed as

Fatty liverGlucagon-like peptide-1 receptor agonistsHepatic fibrosisInsulin resistanceMetabolic associated steatohepatitisMetabolic dysfunction-associated steatotic liver diseaseNon-invasive diagnosticsPersonalized medicineSodium-glucose cotransporter-2 inhibitorsType 2 diabetes mellitus

Identifiers

PMID41696109
PMCPMC12897526

What Socratic holds

Textmetadata
LicenceCC BY-NC
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.