Evidence map›Paper›PMID 41714973›Full record

ArticleBMC infectious diseases2026

Type 2 diabetes mellitus wake-up call: fatal streptococcal toxic shock syndrome triggered by the M1(UK) Streptococcus pyogenes - a case report with genomic insights.

Bishun Deng, Weihong Wen, Ziqi Wang, Qiyong Meng, Haiyang Zhou, Jie Yang, Xingyi Zhu, Yaping Wang, Juan Wang, Zhongwen Zhang and 4 more

Abstract readCase Reports
In one paragraph

Article in BMC infectious diseases, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

0numbers the graph read from it
0cells of the map it votes in
0citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

0 citing papers in PubMed.

No citing paper in PubMed yet.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

14 authors.

Bishun Deng *Department of Laboratory, Guangzhou Medical University Affiliated Qingyuan Hospital (Qingyuan People's Hospital), Guangzhou Medical University, Qing Yuan, 511518, P. R. China.
Weihong Wen *Department of Laboratory, Guangzhou Medical University Affiliated Qingyuan Hospital (Qingyuan People's Hospital), Guangzhou Medical University, Qing Yuan, 511518, P. R. China.
Ziqi Wang *Department of Clinical Laboratory, The First People's Hospital of Foshan (Foshan Hospital Affiliated to Southern University of Science and Technology), School of Medicine, Southern, Foshan, P.R. China.
Qiyong MengDepartment of Emergency Medicine, The Affiliated Qingyuan Hospital (Qingyuan People's Hospital), Guangzhou Medical University, Qing Yuan, P.R. China.
Haiyang ZhouDepartment of Emergency Medicine, The Affiliated Qingyuan Hospital (Qingyuan People's Hospital), Guangzhou Medical University, Qing Yuan, P.R. China.
Jie YangDepartment of Laboratory, Guangzhou Medical University Affiliated Qingyuan Hospital (Qingyuan People's Hospital), Guangzhou Medical University, Qing Yuan, 511518, P. R. China.
Xingyi ZhuDepartment of Laboratory, Guangzhou Medical University Affiliated Qingyuan Hospital (Qingyuan People's Hospital), Guangzhou Medical University, Qing Yuan, 511518, P. R. China.
Yaping WangDepartment of Laboratory, Guangzhou Medical University Affiliated Qingyuan Hospital (Qingyuan People's Hospital), Guangzhou Medical University, Qing Yuan, 511518, P. R. China.
Juan WangDepartment of Laboratory, Guangzhou Medical University Affiliated Qingyuan Hospital (Qingyuan People's Hospital), Guangzhou Medical University, Qing Yuan, 511518, P. R. China.
Zhongwen ZhangDepartment of Laboratory, Guangzhou Medical University Affiliated Qingyuan Hospital (Qingyuan People's Hospital), Guangzhou Medical University, Qing Yuan, 511518, P. R. China.
Yingxian TangDepartment of Laboratory, Guangzhou Medical University Affiliated Qingyuan Hospital (Qingyuan People's Hospital), Guangzhou Medical University, Qing Yuan, 511518, P. R. China.
Lingjuan ChenDepartment of Laboratory, Guangzhou Medical University Affiliated Qingyuan Hospital (Qingyuan People's Hospital), Guangzhou Medical University, Qing Yuan, 511518, P. R. China.
Huihui LuDepartment of Nursing, The Affiliated Qingyuan Hospital (Qingyuan People's Hospital), Guangzhou Medical University, Qing Yuan, 511518, P. R. China. huihui1998@139.com.
Lingqing XuDepartment of Laboratory, Guangzhou Medical University Affiliated Qingyuan Hospital (Qingyuan People's Hospital), Guangzhou Medical University, Qing Yuan, 511518, P. R. China. lingqing_xu@126.com.

Funding

Medical Scientific Research Fund of Qingyuan People's Hospital 202301-201,318Scientific and Technological Research Fund of Guangdong Province for Medical Science and Technology A2021490Scientific Research Grant Fund of Zhong Nanshan Medical Foundation of Guangdong Province ZNSXS-20240010
6 · The paper itself

Abstract

introductionStreptococcus pyogenes (S. pyogenes) can cause streptococcal toxic shock syndrome (STSS), especially in immunocompromised individuals. Early diagnosis is the key to treat STSS effectively. This study presents a fatal case of STSS caused by the emerging M1(UK) S. pyogenes lineage in a patient with type 2 diabetes mellitus (T2DM), alongside a comprehensive literature review. CASE PRESENTATION: A 65-year-old man with T2DM presented with altered consciousness, necrotizing soft tissue injury and was diagnosed with STSS. Bacterial cultures were obtained from right lower limb exudate, right hand mass pus and scrotal pus. Antimicrobial susceptibility testing (AST), whole-genome sequencing (WGS), phylogenomic analysis, and virulence profiling were performed. S. pyogenes isolates from all sites were identified as M1(UK) (emm1/ST28) lineage, closely related to UK strains. The strain was resistant to tetracycline, erythromycin, and clindamycin (ermB, tetM, lmrP) but remained susceptible to β-lactams. Key virulence genes included slo, speC/speG, hasABC and scpA. The slo-mediated endothelial lysis and speC/G-induced cytokine storm contributed to capillary leakage and rapid clinical deterioration. Increased hasABC-mediated capsule expression under hyperglycemia further impaired bacterial clearance.

conclusionThis case highlights the threat of antimicrobial-resistant M1(UK) S. pyogenes in patient with T2DM. The pathogenicity of strain was driven by cytolytic toxins, superantigens, and immune evasion. Early STSS diagnosis is facilitated by microscopic examination and rapid antigen detection test (RADT) of group A streptococcus. CLINICAL TRIAL NUMBER: Not applicable.

Indexed as

Diabetes Mellitus, Type 2Shock, SepticStreptococcal InfectionsStreptococcus pyogenesAgedAnti-Bacterial AgentsFatal OutcomeHumansMaleMicrobial Sensitivity TestsPhylogenyWhole Genome SequencingAnti-Bacterial AgentsEarly diagnosisM1(UK) lineageStreptococcal toxic shock syndromeStreptococcus pyogenesType 2 diabetes mellitus

Identifiers

PMID41714973
PMCPMC13020107

What Socratic holds

Textmetadata
LicenceCC BY-NC-ND
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.