Evidence mapPaperPMID 41724502Full record

Observational studyBMJ open2026

Comparative risk of the neurodegenerative outcomes between sodium-glucose co-transporter 2 (SGLT2) inhibitors and thiazolidinediones in type 2 diabetes: a multicentre cohort study using the Korean healthcare database (2014-2025).

Sang Joon Park, Hye Jeong Kim, Mihae Seo, Dong Won Byun, Kyoil Suh, Myung Hi Yoo, Hyeon Yang, Inho Lee, Soon Hyo Kwon, Minjae Kim and 10 more

Abstract readMulticenter StudyComparative StudyObservational Study
In one paragraph

Observational study in BMJ open, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

0numbers the graph read from it
0cells of the map it votes in
0citing papers in PubMed
field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

0 citing papers in PubMed.

No citing paper in PubMed yet.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

20 authors.

Sang Joon ParkDivision of Endocrinology and Metabolism, Department of Internal Medicine, Soonchunhyang University Hospital Seoul, Yongsan-gu, Korea (the Republic of).ORCID http://orcid.org/0000-0003-2307-6115
Hye Jeong KimDivision of Endocrinology and Metabolism, Department of Internal Medicine, Soonchunhyang University Hospital Seoul, Yongsan-gu, Korea (the Republic of).
Mihae SeoDivision of Endocrinology and Metabolism, Department of Internal Medicine, Soonchunhyang University Hospital Seoul, Yongsan-gu, Korea (the Republic of).
Dong Won ByunDivision of Endocrinology and Metabolism, Department of Internal Medicine, Soonchunhyang University Hospital Seoul, Yongsan-gu, Korea (the Republic of).
Kyoil SuhDivision of Endocrinology and Metabolism, Department of Internal Medicine, Soonchunhyang University Hospital Seoul, Yongsan-gu, Korea (the Republic of).
Myung Hi YooDivision of Endocrinology and Metabolism, Department of Internal Medicine, Soonchunhyang University Hospital Seoul, Yongsan-gu, Korea (the Republic of).
Hyeon YangDepartment of Pediatrics, Soonchunhyang University Hospital Seoul, Yongsan-gu, Korea (the Republic of).
Inho LeeInformatization Project Department, Soonchunhyang University College of Medicine, Seoul, Korea (the Republic of).
Soon Hyo KwonDivision of Nephrology, Department of Internal Medicine, Soonchunhyang University Hospital Seoul, Yongsan-gu, Korea (the Republic of).
Minjae KimDepartment of Psychiatry, Soonchunhyang University Hospital Seoul, Yongsan-gu, Seoul, Korea (the Republic of).ORCID http://orcid.org/0000-0003-4777-7397
Ji-Oh MokDivision of Endocrinology and Metabolism, Department of Internal Medicine, Soon Chun Hyang University Hospital Bucheon, Bucheon-si, Korea (the Republic of).
Dae-Yeon KimDivision of Endocrinology and Metabolism, Department of Internal Medicine, Soonchunhyang University Hospital Cheonan, Cheonan-si, Korea (the Republic of).ORCID http://orcid.org/0000-0003-1715-2062
Seo Young SohnDivision of Endocrinology and Metabolism, Department of Internal Medicine, Myongji Hospital, Goyang-si, Korea (the Republic of).
Rae Woong ParkBiomedical Informatics, Ajou University School of Medicine, Suwon, Korea (the Republic of).ORCID http://orcid.org/0000-0003-4989-3287
Won-Woo SeoInternal Medicine, Kangdong Sacred Heart Hospital, Gangdong-gu, Korea (the Republic of).
So Yoon KwonDivision of Endocrinology and Metabolism, Department of Internal Medicine, Daegu Catholic University Hospital, Daegu, Korea (the Republic of).ORCID http://orcid.org/0000-0002-6282-6612
Sang Youl RheeDepartment of Endocrinology and Metabolism, Kyung Hee University College of Medicine, Dongdaemun-gu, Korea (the Republic of).
Joon-Myoung KwonDepartment of Emergency Medicine, Sejong General Hospital, Bucheon-si, Korea (the Republic of).
Jae Myung ChaDepartment of Gastroenterology, Kyung Hee University Hospital at Gangdong, Gangdong-gu, Korea (the Republic of).
Hyeong Kyu ParkDivision of Endocrinology and Metabolism, Department of Internal Medicine, Soonchunhyang University Hospital Seoul, Yongsan-gu, Korea (the Republic of) hkpark@schmc.ac.kr.

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

objectiveType 2 diabetes mellitus has been associated with an increased risk of cognitive decline and dementia, with patients being 1.5-2 times more likely to develop these conditions. While both sodium-glucose co-transporter 2 (SGLT2) inhibitors and thiazolidinediones (TZDs) have shown potential neuroprotective effects in previous studies, their comparative effectiveness for preventing neurodegenerative outcomes has not been established. This study aimed to compare the risk of stroke, dementia and Alzheimer's disease (AD) between patients treated with SGLT2 inhibitors and those treated with TZDs.

designMulticentre, retrospective, observational, new-user, active-comparator cohort study.

settingElectronic health record-based databases from 11 secondary and tertiary institutions in South Korea from 1 January 2014 to 31 July 2025. The study period began in 2014, following the post-marketing surveillance initiation of SGLT2 inhibitors in Korea (November 2013), to ensure adequate drug availability and clinical adoption.

participantsPatients aged 40 years or older who were newly prescribed either SGLT2 inhibitors or TZDs without prior exposure.

interventionsPropensity score matching (1:1) was performed using sex as the primary covariate due to data availability constraints in the Observational Medical Outcomes Partnership Common Data Model framework. The HRs with 95% CIs were measured via Cox regression analysis.

resultsThe study analysed 24 172 matched pairs for stroke outcomes (40 483 person-years in the SGLT2 inhibitor group and 39 363 person-years in the TZD group), 25 111 matched pairs for dementia (41 924 person-years in the SGLT2 inhibitor group and 40 726 person-years in the TZD group) and 25 237 matched pairs for AD (42 139 person-years in the SGLT2 inhibitor group and 40 895 person-years in the TZD group) across 11 participating hospitals. After a 1:1 propensity score matching, the SGLT2 inhibitors showed no significant difference in stroke risk (HR 1.18, 95% CI 0.62 to 2.23, p=0.62), while having significant reductions in dementia risk (HR 0.66, 95% CI 0.45 to 0.98, p=0.04) and AD risk (HR 0.54, 95% CI 0.35 to 0.83, p=0.005). Moreover, these protective effects for neurodegenerative outcomes were shown to be consistent across multiple hospital sites.

conclusionsSGLT2 inhibitors are associated with a reduced risk of dementia and AD compared with TZDs in patients aged 40 years or older with type 2 diabetes and have neutral effects on stroke risk. These findings confirm the potential selective neuroprotective benefits of SGLT2 inhibitors for neurodegenerative outcomes, which may inform therapeutic decision-making for diabetic patients at risk of cognitive decline.

Indexed as

Alzheimer DiseaseDementiaDiabetes Mellitus, Type 2Hypoglycemic AgentsSodium-Glucose Transporter 2 InhibitorsStrokeThiazolidinedionesAgedDatabases, FactualFemaleHumansMaleMiddle AgedRepublic of KoreaRetrospective StudiesHypoglycemic AgentsSodium-Glucose Transporter 2 InhibitorsThiazolidinedionesDementiaHealth informaticsStroke

Identifiers

PMID41724502
PMCPMC12927369

What Socratic holds

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Registered trials

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.