Evidence mapPaperPMID 41744888Full record

ReviewCurrent oncology (Toronto, Ont.)2026

Endocrine Therapy for Endometrial Carcinoma: Current Evidence, Resistance Mechanisms, and Biomarker-Driven Patient Selection.

Taro Yamanaka, Hiroshi Yoshida, Tatsunori Shimoi, Kazuki Sudo, Kan Yonemori

Abstract readReview
In one paragraph

Review in Current oncology (Toronto, Ont.), 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 2 papers.

0numbers the graph read from it
0cells of the map it votes in
2citing papers in PubMed
field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

2 citing papers in PubMed.

  1. Review
  2. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

5 authors.

Taro YamanakaDepartment of Medical Oncology, National Cancer Center Hospital, Tokyo 104-0045, Japan.ORCID 0000-0001-9346-5090
Hiroshi YoshidaDepartment of Diagnostic Pathology, National Cancer Center Hospital, Tokyo 104-0045, Japan.ORCID 0000-0002-7569-7813
Tatsunori ShimoiDepartment of Medical Oncology, National Cancer Center Hospital, Tokyo 104-0045, Japan.ORCID 0000-0002-3603-8575
Kazuki SudoDepartment of Medical Oncology, National Cancer Center Hospital, Tokyo 104-0045, Japan.
Kan YonemoriDepartment of Medical Oncology, National Cancer Center Hospital, Tokyo 104-0045, Japan.

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

The treatment landscape for endometrial carcinoma (EC) is undergoing a paradigm shift from traditional histopathological dualism to precision medicine grounded in the Cancer Genome Atlas (TCGA) molecular classification. The "No Specific Molecular Profile" (NSMP) subgroup, the largest molecular cohort, has emerged as a particularly promising target for endocrine-based strategies. While endocrine therapy (ET) has been a mainstay for over 60 years due to its favorable safety profile, its efficacy as monotherapy remains modest. This review provides a comprehensive overview of current endocrine strategies, including traditional agents like progestins and aromatase inhibitors, and focuses on novel combination therapies designed to overcome resistance. Recent clinical trials have demonstrated that integrating molecularly targeted agents, such as CDK4/6 and mTOR inhibitors, significantly improves clinical outcomes. Specifically, patients with

Indexed as

Antineoplastic Agents, HormonalEndometrial NeoplasmsBiomarkers, TumorDrug Resistance, NeoplasmFemaleHumansPatient SelectionAntineoplastic Agents, HormonalBiomarkers, TumorCDK4/6 inhibitorendocrine therapyendometrial cancerestrogen receptormTOR inhibitorprogesterone receptorprogestinselective estrogen receptor degraders

Identifiers

PMID41744888
PMCPMC12939215

What Socratic holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.