Evidence mapPaperPMID 41745976Full record

ReviewVeterinary sciences2026

The Collaborative Collapse: Bile Acid Dysmetabolism as a Central Pathogenic Driver in Canine and Feline Multi-Systemic Disorders-From Mechanisms to Precision Therapeutics.

Krisztián Németh, István Tóth, Katalin Lányi, Boglárka Mária Schilling-Tóth, Szilveszter Csorba, Ivona Žura Žaja, Ágnes Sterczer

Abstract readReview
In one paragraph

Review in Veterinary sciences, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 1 paper.

0numbers the graph read from it
0cells of the map it votes in
1citing papers in PubMed
field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

1 citing paper in PubMed.

  1. Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

7 authors.

Krisztián NémethDepartment of Physiology and Biochemistry, University of Veterinary Medicine Budapest, István u. 2, H-1078 Budapest, Hungary.ORCID 0009-0006-2470-4792
István TóthDepartment of Physiology and Biochemistry, University of Veterinary Medicine Budapest, István u. 2, H-1078 Budapest, Hungary.ORCID 0000-0002-0168-4753
Katalin LányiFood Chain Laboratory Center, Institute of Food Chain Science, University of Veterinary Medicine Budapest, Marek J. u. 2, H-1078 Budapest, Hungary.ORCID 0000-0003-1992-6324
Boglárka Mária Schilling-TóthDepartment of Physiology and Biochemistry, University of Veterinary Medicine Budapest, István u. 2, H-1078 Budapest, Hungary.
Szilveszter CsorbaDepartment of Digital Food Science, Institute of Food Chain Science, University of Veterinary Medicine Budapest, István u. 2, H-1078 Budapest, Hungary.ORCID 0000-0002-2238-4469
Ivona Žura ŽajaDepartment of Physiology and Radiobiology, Faculty of Veterinary Medicine, University of Zagreb, Heinzelova 55, 10000 Zagreb, Croatia.ORCID 0000-0002-0595-2524
Ágnes SterczerDepartment of Internal Medicine, University of Veterinary Medicine Budapest, István u. 2, H-1078 Budapest, Hungary.

Funding

Ministry of Culture and Innovation of Hungary from the National Research, Development and Innovation Fund 2025-2.1.1-EKÖP-2025-00022University of Veterinary Medicine Budapest SRF-003
6 · The paper itself

Abstract

Veterinary metabolomics has redefined bile acids (BAs) from simple digestive surfactants to systemic endocrine signals within a microbial-host metabolic axis. This review aims to evaluate how BA dysmetabolism acts as a central pathogenic factor in canine and feline disease. We analyze the BA pool's integrity, which depends on a specialized functional guild, primarily

Indexed as

bile acidschronic enteropathyextracellular vesiclesGut–X axisMCBAmicrobiomePeptacetobacter hiranonispostbioticsSynComsTGR5

Identifiers

PMID41745976
PMCPMC12944954

What Socratic holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.