Evidence map›Paper›PMID 41750369›Full record

ReviewBiomolecules2026

ELABELA: A Potential Therapeutic Target for Ischemia-Reperfusion Injury.

Wan Li, Cuicui Meng, Shujun Yang, Yibei Xie, Zijie Huang, Tong Wang

Abstract readReview
In one paragraph

Review in Biomolecules, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 1 paper.

0numbers the graph read from it
0cells of the map it votes in
1citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

1 citing paper in PubMed.

  1. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

6 authors.

Wan LiDepartment of Emergency, The Eighth Affiliated Hospital of Sun Yat-sen University, Shenzhen 518033, China.
Cuicui MengDepartment of Emergency, The Eighth Affiliated Hospital of Sun Yat-sen University, Shenzhen 518033, China.
Shujun YangDepartment of Emergency, The Eighth Affiliated Hospital of Sun Yat-sen University, Shenzhen 518033, China.
Yibei XieDepartment of Emergency, The Eighth Affiliated Hospital of Sun Yat-sen University, Shenzhen 518033, China.
Zijie HuangDepartment of Emergency, The Eighth Affiliated Hospital of Sun Yat-sen University, Shenzhen 518033, China.
Tong WangDepartment of Emergency, The Eighth Affiliated Hospital of Sun Yat-sen University, Shenzhen 518033, China.

Funding

Guangdong Basic and Applied Basic Research Foundation 2023A1515220199, 2024A1515220088Sanming Project of Medicine in Shenzhen SZSM202402020the Fundamental Research Funds for the Central Universities 13ykzd16, 17ykjc18the Futian District Health and Public Welfare Research Project of Shenzhen City FTWS2019001, FTWS2021016, FTWS2022018, FTWS2023064the National Natural Science Foundation of China 81070125, 81270213, 81670306the National Natural Science Foundation of Guangdong Province 2017A030313503the Science and Technology Foundation in Guangdong Province 2014A020211002the Science and Technology Foundation in Guangzhou City 201806020084the Shenzhen Science and Technology Program JCYJ20190808101405466, JCYJ20210324115003008, JCYJ20220530144404009, KCXFZ20230731094100002
6 · The paper itself

Abstract

Ischemia-reperfusion injury (IRI) refers to a paradoxical pathological process in which ischemic tissues or organs sustain further damage upon the restoration of blood flow and is clinically commonly observed in the peripheral vascular systems and various vital organs. In 2013, the endogenous peptidic hormone ELABELA (ELA) was identified as a novel native ligand of the APJ G protein-coupled receptor, alongside apelin. ELA is constitutively expressed in a range of adult and embryonic tissues and plays a role in numerous physiological and pathological processes. In recent years, a rapidly growing body of research has highlighted the cytoprotective properties of ELA against IRI. In this review, we synthesize current research on the protective role of ELA in mitigating IRI across multiple organ systems.

Indexed as

Peptide HormonesReperfusion InjuryAnimalsApoptosisHumansOxidative StressAPELA protein, humanPeptide HormonesangiogenesisapoptosisELABELAfibrosisinflammatory responseischemia–reperfusion injuryoxidative stress

Identifiers

PMID41750369
PMCPMC12937643

What Socratic holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.