Evidence map›Paper›PMID 41758899›Full record

ArticlePLoS pathogens2026

Plasma albumin contributes to early Yersinia pestis survival at the onset of flea infection.

Amélie Dewitte, Florent Sebbane, Sébastien Bontemps-Gallo

Abstract read
In one paragraph

Article in PLoS pathogens, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 1 paper.

0numbers the graph read from it
0cells of the map it votes in
1citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

1 citing paper in PubMed.

  1. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

3 authors.

Amélie DewitteUniv. Lille, CNRS, Inserm, CHU Lille, Institut Pasteur de Lille, U1019 - UMR 9017 - CIIL - Center for Infection and Immunity of Lille, Lille, France.
Florent SebbaneUniv. Lille, CNRS, Inserm, CHU Lille, Institut Pasteur de Lille, U1019 - UMR 9017 - CIIL - Center for Infection and Immunity of Lille, Lille, France.
Sébastien Bontemps-GalloUniv. Lille, CNRS, Inserm, CHU Lille, Institut Pasteur de Lille, U1019 - UMR 9017 - CIIL - Center for Infection and Immunity of Lille, Lille, France.ORCID https://orcid.org/0000-0002-1834-5416

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

The plague bacillus, Yersinia pestis, encounters a hostile oxidative environment upon entering the flea gut, a barrier it must overcome to initiate biofilm formation for transmission. This resilience has been attributed mainly to bacterial defenses, yet we show that host plasma proteins act as transient allies, consistent with a role in mitigating oxidative stress immediately after the blood meal. These findings reframe blood not only as nutrition but also as an active modulator of vector competence.

Indexed as

PlagueSiphonapteraYersinia pestisAnimalsInsect VectorsOxidative Stress

Identifiers

PMID41758899
PMCPMC12962530

What Socratic holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.