Evidence mapPaperPMID 41769065Full record

ArticleBiochemistry and biophysics reports2026

The miR-2110/

ThanhLoan Tran, Zhong-Yu Wang, Pei-Shan Li, Ying Yang, Yi-Wei Zhang, Shu-Ming Zhang, PhongSon Dinh, NgocLong Le, TrungHieu Pham, Ling Huang and 3 more

Abstract read
In one paragraph

Article in Biochemistry and biophysics reports, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

0numbers the graph read from it
0cells of the map it votes in
0citing papers in PubMed
field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

0 citing papers in PubMed.

No citing paper in PubMed yet.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

13 authors.

ThanhLoan TranDepartment of Pathophysiology, Guangxi Medical University, Nanning, Guangxi, 530021, China.
Zhong-Yu WangDepartment of Pathophysiology, Guangxi Medical University, Nanning, Guangxi, 530021, China.
Pei-Shan LiDepartment of Pathophysiology, Guangxi Medical University, Nanning, Guangxi, 530021, China.
Ying YangDepartment of Pathophysiology, Guangxi Medical University, Nanning, Guangxi, 530021, China.
Yi-Wei ZhangDepartment of Pathophysiology, Guangxi Medical University, Nanning, Guangxi, 530021, China.
Shu-Ming ZhangDepartment of Pathophysiology, Guangxi Medical University, Nanning, Guangxi, 530021, China.
PhongSon DinhCollege of Medicine and Pharmacy, Duy Tan University, Da Nang, 550000, Viet Nam.
NgocLong LeDepartment of General Medicine, Binh Duong General Hospital, Binh Duong, 820000, Viet Nam.
TrungHieu PhamDepartment of Urology, Binh Duong General Hospital, Binh Duong, 820000, Viet Nam.
Ling HuangDepartment of Pathophysiology, Guangxi Medical University, Nanning, Guangxi, 530021, China.
Ning-Yuan ChenDepartment of Pathophysiology, Guangxi Medical University, Nanning, Guangxi, 530021, China.
Jun-Hua PengDepartment of Pathophysiology, Guangxi Medical University, Nanning, Guangxi, 530021, China.
Shang-Ling PanDepartment of Pathophysiology, Guangxi Medical University, Nanning, Guangxi, 530021, China.

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Coronary heart disease (CHD) is driven by endothelial dysfunction and chronic vascular inflammation. hsa-miR-2110 (miR-2110) has been associated with adverse cardiovascular outcomes, but its mechanistic role in CHD remains unclear. In this study, miR-2110 expression was quantified in peripheral blood from CHD patients and healthy controls. Functional effects were assessed in EA.hy926 endothelial cells following lentiviral overexpression of miR-2110. The target gene

Indexed as

AtherosclerosisCoronary heart diseaseEndothelial dysfunctionInflammatory signalingmiR-2110TRAF3

Identifiers

PMID41769065
PMCPMC12937024

What Socratic holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.