Evidence mapPaperPMID 41782138Full record

ReviewJournal of translational medicine2026

The role of histone methylation in fibrosis and its therapeutic potential.

Zaixiao Tao, Hao Zhang, Pengfei Zuo, Lingdi Hua

Abstract readReview
In one paragraph

Review in Journal of translational medicine, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 1 paper.

0numbers the graph read from it
0cells of the map it votes in
1citing papers in PubMed
field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

1 citing paper in PubMed.

  1. Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

4 authors.

Zaixiao Tao *Department of Cardiology, The Second Affiliated Hospital of Nanjing Medical University, No. 121 Jiangjiayuan, Nanjing, 210011, China.
Hao Zhang *Department of Cardiology, The Second Affiliated Hospital of Nanjing Medical University, No. 121 Jiangjiayuan, Nanjing, 210011, China.
Pengfei ZuoDepartment of Cardiology, Zhongda Hospital, Southeast University, Nanjing, Jiangsu, China. zuopengfei1207@sina.com.
Lingdi HuaDepartment of Cardiology, Nanjing Yuhua Hospital, No.9, Banqiao Avenue, Yuhuatai District, Nanjing, 210039, China. yiyih0328@sina.com.

Funding

Medical Scientific Research Project of Jiangsu Provincial Health Commission M2024018Nanjing City Development Project of Medical Science and Technology YKK23267
6 · The paper itself

Abstract

backgroundFibrosis is a pathological process characterized by excessive extracellular matrix deposition, leading to tissue stiffening and organ dysfunction, posing a significant threat to human health. In recent years, histone methylation, a key epigenetic modification, has been increasingly recognized for its crucial regulatory role in the initiation and progression of fibrosis. By modulating chromatin structure and gene expression, histone methylation influences the activation of fibrosis-related cells, inflammatory responses, and cellular fate transitions. MAIN BODY: This review systematically summarizes the molecular mechanisms of histone methylation in fibrosis across multiple organs, including the kidney, liver, heart, and lungs. It emphasizes the functions and regulatory networks of methyltransferases and demethylases, integrating the latest experimental and clinical findings. Furthermore, potential therapeutic strategies targeting histone methylation are discussed, highlighting their promise for precise anti-fibrotic interventions.

conclusionsBy consolidating advances in multi-organ fibrosis research, this article aims to provide a theoretical foundation and practical guidance for the future development of targeted fibrosis therapies.

Indexed as

FibrosisHistonesAnimalsEpigenesis, GeneticHumansMethylationHistonesDemethylaseEpigeneticsFibrosisHistone methylationMethyltransferaseTherapeutic strategies

Identifiers

PMID41782138
PMCPMC13067678

What Socratic holds

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LicenceCC BY-NC-ND
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Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.