Evidence map›Paper›PMID 41795808›Full record

ArticleThe Journal of international medical research2026

Clinical characterization of 39 patients with autoimmune pulmonary alveolar proteinosis.

Yan-Fen Zhong, Xiao Lv

Abstract read
In one paragraph

Article in The Journal of international medical research, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

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0citing papers in PubMed
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1 · What the graph read from it

What it found

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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

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Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

0 citing papers in PubMed.

No citing paper in PubMed yet.

4 · The record

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PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

2 authors.

Yan-Fen ZhongDepartment of Respiratory Medicine, Fuzhou Pulmonary Hospital, China.ORCID 0009-0009-8348-3469
Xiao LvDepartment of Respiratory Medicine, Fuzhou Pulmonary Hospital, China.

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

ObjectivesWe aimed to explore the clinical manifestations, examination findings, diagnostic methods, and treatment efficacy of patients with pulmonary alveolar proteinosis and improve clinicians' understanding of this disease.MethodsWe retrospectively analyzed the clinical characteristics of 39 patients diagnosed with autoimmune pulmonary alveolar proteinosis admitted to Fuzhou Pulmonary Hospital, Fujian Province, from August 2013 to December 2024. We also analyzed the correlations between lactate dehydrogenase, arterial partial oxygen pressure, and pulmonary alveolar arterial oxygen differential pressure, lung function, tumor markers, blood lipids, and disease severity. Furthermore, the relevant literature was reviewed.ResultsOf the 39 patients, 23 were male, 16 were female, 18 were smokers, and 11 had pneumoconiosis. The main clinical symptoms were cough and shortness of breath. The typical manifestations of chest computed tomography were "map-like" changes or "crazy-paving pattern". The main manifestations of pulmonary function were diffusion dysfunction and restricted ventilation dysfunction. pulmonary alveolar proteinosis disease severity score was positively correlated with lactate dehydrogenase, pulmonary alveolar arterial oxygen differential pressure and the tumor markers carcinoembryonic antigen, cytokeratin 19 fragment antigen 21-1, neuron specific enolase, and carbohydrate antigen 153 (

Indexed as

Autoimmune DiseasesPulmonary Alveolar ProteinosisAdultAgedBiomarkersFemaleHumansL-Lactate DehydrogenaseLungMaleMiddle AgedOxygenRespiratory Function TestsRetrospective StudiesSeverity of Illness IndexTomography, X-Ray ComputedBiomarkersL-Lactate DehydrogenaseOxygenarterial partial oxygen pressurecarbon monoxide dispersion to the predicted valuedisease severity scorelactate dehydrogenasepulmonary alveolar arterial oxygen differential pressurePulmonary alveolar proteinosistumor marker

Identifiers

PMID41795808
PMCPMC12968407

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.