Trial reportExperimental physiology2026
Report results: Impact of whole-body resistance exercise timing on mitigating hyperglycaemia-induced vascular dysfunction in healthy adults.
Trial report in Experimental physiology, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 1 paper.
What it found
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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
The trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.
Who cites it
1 citing paper in PubMed.
- Report results: Impact of whole-body resistance exercise timing on mitigating hyperglycaemia-induced vascular dysfunction in healthy adults.Experimental physiology · 2026Trial
Corrections and comments
PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.
Authors and funding
7 authors.
Funding
Abstract
Postprandial hyperglycaemia is characterized by elevated blood glucose levels following a meal and is associated with impaired vascular endothelial function. Regular exercise has been shown to preserve and improve vascular endothelial function. However, whether there is an optimal time to exercise to mitigate postprandial hyperglycaemia-related vascular dysfunction is unknown. This randomized crossover study recruited healthy adults to compare four exercise timing conditions with a non-exercise Control. Participants performed whole-body resistance exercises at one of 30 min pre (30Pre), immediately post (IP), 30 min post (30Post), or 60 min post (60Post) consuming a high carbohydrate meal. Measures of blood glucose and endothelial function (via flow-mediated dilation of the brachial artery) were assessed at fasting and incrementally to 120 min following the meal. Exercise performed IP improved flow-mediated dilation by 2.3 ± 2.7% (P = 0.04) and lowered postprandial glucose by 1.5 ± 1.8 mmol/L (P < 0.01) at 1 h postprandially, compared to the Control. 30Post and 60post significantly decreased glucose post exercise by 1.06 ± 2.26 mmol/L (P = 0.002) compared to the control. Comparatively, pre-meal exercise did not improve postprandial vascular function or glucose compared to the control or other timing conditions. These findings add precision to the existing literature on postprandial exercise by identifying immediately after eating as most effective for improving vascular and metabolic outcomes relevant to cardiometabolic disease prevention.
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Registered trials
Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.