ArticleJournal of inflammation research2026
Loss of Potassium and Chloride Transport Changes PM-Induced Epithelial Dysfunction.
Article in Journal of inflammation research, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.
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Abstract
Background: Chronic exposure to particulate matter (PM) is recognized as a significant contributor to respiratory health complications, including oxidative stress, inflammatory responses, and compromised epithelial barrier function. In this work, we ask whether the transport of potassium and chloride through the large-conductance calcium-activated potassium (BK Methods: This study aimed to evaluate the impact of PM on cell variability, ROS level, inflammation, mitochondrial function, intracellular calcium homeostasis, and epithelial barrier integrity in three different airway epithelial cell lines: wild-type human bronchial epithelial cells (HBE WT), HBE WT cells with disruption of the Results: PM exposure significantly increased ROS synthesis and amplified IL-6 and TNF-α release, particularly in HBE ΔαBK Conclusion: These findings indicate that impaired potassium and chloride transport through the BK
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