ArticleFunction (Oxford, England)2026
Dietary salt impairs circadian physiological metabolic adaptations in salt-sensitive hypertension.
Lashodya V Dissanayake, Adrian Zietara, Ratnakar Tiwari, Melissa Lowe, Olha Kravtsova, Vladislav Levchenko, Ruslan Bohovyk, Michelle L Gumz, Alexander Staruschenko
Abstract read
In one paragraphArticle in Function (Oxford, England), 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.
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What it found
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2 · The registryThe trial behind it
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4 · The recordCorrections and comments
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5 · Who and what moneyAuthors and funding
9 authors.
Lashodya V DissanayakeDepartment of Molecular Pharmacology & Physiology, Morsani College of Medicine, University of South Florida, Tampa, Florida, United States.ORCID 0000-0002-2329-0931 Adrian ZietaraDepartment of Molecular Pharmacology & Physiology, Morsani College of Medicine, University of South Florida, Tampa, Florida, United States.
Ratnakar TiwariDepartment of Molecular Pharmacology & Physiology, Morsani College of Medicine, University of South Florida, Tampa, Florida, United States.
Melissa LoweDepartment of Molecular Pharmacology & Physiology, Morsani College of Medicine, University of South Florida, Tampa, Florida, United States.
Olha KravtsovaDepartment of Molecular Pharmacology & Physiology, Morsani College of Medicine, University of South Florida, Tampa, Florida, United States.
Vladislav LevchenkoDepartment of Molecular Pharmacology & Physiology, Morsani College of Medicine, University of South Florida, Tampa, Florida, United States.
Ruslan BohovykDepartment of Molecular Pharmacology & Physiology, Morsani College of Medicine, University of South Florida, Tampa, Florida, United States.
Michelle L GumzDepartment of Physiology and Aging, University of Florida, Gainesville, Florida, United States.
Alexander StaruschenkoDepartment of Molecular Pharmacology & Physiology, Morsani College of Medicine, University of South Florida, Tampa, Florida, United States.ORCID 0000-0002-5190-8356 Funding
Training in Research on Vascular Inflammation and InjuryT32HL160529 · NHLBI · UNIVERSITY OF SOUTH FLORIDA · 2022 to 2025
$688kSexual dimorphisms and role of the cGAS-STING pathway in diabetic nephropathyR01DK135644 · UNIVERSITY OF SOUTH FLORIDA · 2025 to 2025
$513kRenal diabetic complications mediated by the PAR1 signaling in podocytesR01DK129227 · UNIVERSITY OF SOUTH FLORIDA · 2025 to 2025
$326kAmerican Heart Association 25POST1374965American Heart Association 25POST1375066BLRD VA I01 BX004024NHLBI NIH HHS T32 HL160529NIDDK NIH HHS R01 DK129227NIDDK NIH HHS R01 DK135644NIH HHS R01 DK109570NIH HHS R01 DK129227NIH HHS R01 DK135644NIH HHS T32 HL160529U.S. Department of Veteran Affairs IK6 RD001204USF Hypertension and Kidney Research Center Early Stage Investigator GrantUSF Hypertension and Kidney Research Center Multi-PI Grant
6 · The paper itselfAbstract
The body's circadian rhythm is coordinated by core clock proteins [period (PER), cryptochrome circadian regulator (CRY), circadian locomotor output cycles kaput (CLOCK), and basic helix-loop-helix ARNT-like protein 1 (BMAL1)] that function in both the central hypothalamic and peripheral tissue molecular clocks. Our recent study demonstrated that deletion of
Indexed as
Adaptation, PhysiologicalCircadian RhythmHypertensionSodium Chloride, DietaryAnimalsBlood PressureKidneyMalePeriod Circadian ProteinsProteomeRatsRats, Inbred DahlTranscriptomePer1 protein, ratPeriod Circadian ProteinsProteomeSodium Chloride, Dietaryclock genesDahl salt-sensitive ratdiurnal variationkidney transcriptomicsPER1
Identifiers
PMID41811151
PMCPMC13053008
What Socratic holds
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