Evidence mapPaperPMID 41822029Full record

ReviewFrontiers in cell and developmental biology2025

Multidimensional mechanisms of quercetin in diabetic kidney disease.

Zhaoyuan Gong, Qianzi Che, Chuxuan Wang, Bin Liu, Mingzhi Hu, Haili Zhang, Ning Liang, Huizhen Li, Guozhen Zhao, Lijiao Yan and 4 more

Abstract readReview
In one paragraph

Review in Frontiers in cell and developmental biology, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 3 papers.

0numbers the graph read from it
0cells of the map it votes in
3citing papers in PubMed
field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

3 citing papers in PubMed.

  1. Article
  2. Review
  3. Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

14 authors.

Zhaoyuan Gong *Institute of Basic Research in Clinical Medicine, China Academy of Chinese Medical Sciences, Beijing, China.
Qianzi Che *Institute of Basic Research in Clinical Medicine, China Academy of Chinese Medical Sciences, Beijing, China.
Chuxuan Wang *Beijing University of Chinese Medicine, Beijing, China.
Bin Liu *Institute of Basic Research in Clinical Medicine, China Academy of Chinese Medical Sciences, Beijing, China.
Mingzhi HuInstitute of Basic Research in Clinical Medicine, China Academy of Chinese Medical Sciences, Beijing, China.
Haili ZhangInstitute of Basic Research in Clinical Medicine, China Academy of Chinese Medical Sciences, Beijing, China.
Ning LiangInstitute of Basic Research in Clinical Medicine, China Academy of Chinese Medical Sciences, Beijing, China.
Huizhen LiInstitute of Basic Research in Clinical Medicine, China Academy of Chinese Medical Sciences, Beijing, China.
Guozhen ZhaoInstitute of Basic Research in Clinical Medicine, China Academy of Chinese Medical Sciences, Beijing, China.
Lijiao YanInstitute of Basic Research in Clinical Medicine, China Academy of Chinese Medical Sciences, Beijing, China.
Tian SongInstitute of Basic Research in Clinical Medicine, China Academy of Chinese Medical Sciences, Beijing, China.
Lin ChenInstitute of Basic Research in Clinical Medicine, China Academy of Chinese Medical Sciences, Beijing, China.
Nannan ShiInstitute of Basic Research in Clinical Medicine, China Academy of Chinese Medical Sciences, Beijing, China.
Jing GuoInstitute of Basic Research in Clinical Medicine, China Academy of Chinese Medical Sciences, Beijing, China.

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Quercetin is a flavonoid compound that has demonstrated substantial potential in the treatment of diabetic kidney disease (DKD). However, there is still a lack of systematic research on the exact mechanism of action of quercetin. This review discusses the druggability, molecular targets, and signaling pathways of quercetin in DKD treatment. We retrieved the latest research on the pharmacological effects and mechanisms related to quercetin from PubMed and Scopus as of June 2025 (2012-2025). Evidence suggests that quercetin has the potential to eliminate senescent cells in DKD. Network pharmacology was used to predict the targets and pathways of quercetin in targeting cellular senescence to treat DKD. Using on existing research, it was further confirmed that quercetin can effectively act on hub target and pathway. The mechanism of quercetin therapy in DKD was summarized from three dimensions: inflammation, oxidative stress, and cell death. This review highlights the potential of quercetin for treating DKD by providing a biological basis for its mechanism of action and its use as a senolytic drug for this disease.

Indexed as

cellular senescencediabetic kidney diseaseliterature reviewnetwork pharmacologyquercetin

Identifiers

PMID41822029
PMCPMC12975762

What Socratic holds

Textmetadata
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Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.