ArticleExperimental and therapeutic medicine2026
Effects of the WNT signaling pathway on inflammation and fibrosis in idiopathic pulmonary fibrosis: Clinical, radiological and molecular evaluation.
Article in Experimental and therapeutic medicine, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.
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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
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Abstract
Idiopathic pulmonary fibrosis (IPF) is a progressive and fatal interstitial lung disease characterized by irreversible fibrosis, radiological honeycombing and a progressive decline in pulmonary function. Although antifibrotic agents such as pirfenidone and nintedanib can slow disease progression, these agents fail to reverse established structural damage, underscoring the urgent need for novel therapeutic strategies. Notably, the WNT signaling pathway has been implicated in fibrogenesis, suggesting it may represent a promising therapeutic target in IPF. The present study aimed to elucidate the role of WNT signaling in IPF pathogenesis, and to evaluate the antifibrotic and anti-inflammatory effects of the WNT inhibitors ETC-159 and LGK-974
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