Evidence mapPaperPMID 41830757Full record

ArticleThe journal of prevention of Alzheimer's disease2026

Living arrangements and cognitive resilience in aging: unraveling distinct pathways through plasma biomarkers.

Yuanyuan Peng, Heqianxi Dong, Yu Luo, Wen Zhou, Lu Liu, Ming Chen, Na Liu, Jiwen Che, Feifei Hu, Yifeng Cheng and 2 more

Abstract read
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Article in The journal of prevention of Alzheimer's disease, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

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1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

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Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

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4 · The record

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PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

12 authors.

Yuanyuan PengHubei Provincial Clinical Research Center for Alzheimer's Disease, Tianyou Hospital Affiliated to Wuhan University of Science and Technology, Brain Science and Advanced Technology Institute, Wuhan University of Science and Technology, Wuhan, Hubei 430065, China.
Heqianxi DongHubei Provincial Clinical Research Center for Alzheimer's Disease, Tianyou Hospital Affiliated to Wuhan University of Science and Technology, Brain Science and Advanced Technology Institute, Wuhan University of Science and Technology, Wuhan, Hubei 430065, China.
Yu LuoHubei Provincial Clinical Research Center for Alzheimer's Disease, Tianyou Hospital Affiliated to Wuhan University of Science and Technology, Brain Science and Advanced Technology Institute, Wuhan University of Science and Technology, Wuhan, Hubei 430065, China.
Wen ZhouHubei Provincial Clinical Research Center for Alzheimer's Disease, Tianyou Hospital Affiliated to Wuhan University of Science and Technology, Brain Science and Advanced Technology Institute, Wuhan University of Science and Technology, Wuhan, Hubei 430065, China.
Lu LiuHubei Provincial Clinical Research Center for Alzheimer's Disease, Tianyou Hospital Affiliated to Wuhan University of Science and Technology, Brain Science and Advanced Technology Institute, Wuhan University of Science and Technology, Wuhan, Hubei 430065, China.
Ming ChenHubei Provincial Clinical Research Center for Alzheimer's Disease, Tianyou Hospital Affiliated to Wuhan University of Science and Technology, Brain Science and Advanced Technology Institute, Wuhan University of Science and Technology, Wuhan, Hubei 430065, China.
Na LiuHubei Provincial Clinical Research Center for Alzheimer's Disease, Tianyou Hospital Affiliated to Wuhan University of Science and Technology, Brain Science and Advanced Technology Institute, Wuhan University of Science and Technology, Wuhan, Hubei 430065, China.
Jiwen CheHubei Provincial Clinical Research Center for Alzheimer's Disease, Tianyou Hospital Affiliated to Wuhan University of Science and Technology, Brain Science and Advanced Technology Institute, Wuhan University of Science and Technology, Wuhan, Hubei 430065, China.
Feifei HuHubei Provincial Clinical Research Center for Alzheimer's Disease, Tianyou Hospital Affiliated to Wuhan University of Science and Technology, Brain Science and Advanced Technology Institute, Wuhan University of Science and Technology, Wuhan, Hubei 430065, China.
Yifeng ChengHubei Provincial Clinical Research Center for Alzheimer's Disease, Tianyou Hospital Affiliated to Wuhan University of Science and Technology, Brain Science and Advanced Technology Institute, Wuhan University of Science and Technology, Wuhan, Hubei 430065, China.
Xinyan XieHubei Provincial Clinical Research Center for Alzheimer's Disease, Tianyou Hospital Affiliated to Wuhan University of Science and Technology, Brain Science and Advanced Technology Institute, Wuhan University of Science and Technology, Wuhan, Hubei 430065, China; School of Public Health, Wuhan University of Science and Technology, Wuhan 430065, China. Electronic address: xiexinyan@wust.edu.cn.
Yan ZengHubei Provincial Clinical Research Center for Alzheimer's Disease, Tianyou Hospital Affiliated to Wuhan University of Science and Technology, Brain Science and Advanced Technology Institute, Wuhan University of Science and Technology, Wuhan, Hubei 430065, China. Electronic address: zengyan68@wust.edu.cn.

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

backgroundGlobal aging and changing family structures necessitate identifying modifiable factors for cognitive health. While social isolation is a known risk, the protective role of specific living arrangements and their interplay with neurobiology is unclear.

objectivesThis study aimed to: (1) examine the longitudinal association between living arrangements and cognitive function in older adults, and (2) investigate the potential moderating roles of plasma Alzheimer's disease (AD) biomarkers in this relationship.

methodsUsing data from the Hubei Memory and Aging Cohort Study, we followed 3403 older adults aged 65 years and above with different living arrangements. Participants underwent standardized cognitive assessments and plasma biomarker measurements, including amyloid-beta (Abeta) 40, Abeta 42, glial fibrillary acidic protein (GFAP), neurofilament light chain (NfL), phosphorylated tau (p-tau) 181, and p-tau 217. Linear mixed-effects models were employed to analyze cognitive trajectories.

resultsCompared to older adults living separately from their families, those in two specific living arrangements, living with a spouse only or in multigenerational living, demonstrated significantly better cognitive performance across multiple domains. These protective associations proved robust even after comprehensive adjustment for plasma AD biomarkers. Importantly, we found that higher plasma GFAP levels significantly attenuated the cognitive benefits conferred by favorable living arrangements. In a separate, distinct pathway, higher plasma Abeta40 levels were independently associated with better-preserved language function over time.

conclusionsFavorable living arrangements may benefit cognitive health through pathways independent of typical AD pathology. Incorporating living arrangements and plasma biomarkers, particularly GFAP, could enhance risk assessment and targeted interventions for cognitive decline in older adults.

Indexed as

AgingCognitionAgedAged, 80 and overAlzheimer DiseaseAmyloid beta-PeptidesBiomarkersFamily StructureFemaleGlial Fibrillary Acidic ProteinHumansLongitudinal StudiesMaleNeurofilament ProteinsPeptide Fragmentstau ProteinsAmyloid beta-PeptidesBiomarkersGlial Fibrillary Acidic ProteinNeurofilament ProteinsPeptide Fragmentstau ProteinsCohort studyDomain-specific cognitionLiving arrangementsOlder adultsPlasma AD biomarkers

Identifiers

PMID41830757
PMCPMC12996944

What Socratic holds

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LicenceCC BY-NC-ND
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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.