ArticleFrontiers in nutrition2026
Article in Frontiers in nutrition, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.
What it found
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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
The trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.
Who cites it
0 citing papers in PubMed.
No citing paper in PubMed yet.
Corrections and comments
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Authors and funding
9 authors.
Funding
No grant is acknowledged in the PubMed record.
Abstract
Background: Obesity is closely linked to gut microbiota dysbiosis and metabolic disorders. Methods: C57BL/6J mice were fed an HFD supplemented with 3.2% POP for 17 weeks. Obesity-related parameters, gut microbiota, and serum metabolomics were analyzed. Results: POP significantly reduced obesity, improved lipid profiles and glucose homeostasis, increased gut microbiota diversity, and normalized the Firmicutes/Bacteroidetes ratio. It modulated several key gut microbiota genera and altered metabolites including LacCer (d18:1/12:0) and N-(4,7-Dihydroxy-8-Methyl-2-Oxo-2H-Chromen-3-Yl)-2,2-Dimethylchromane-6-Carboxamide (NDC), which strongly correlated with obesity-related indices. Conclusion: POP may improve HFD-induced obesity by regulating gut microbiota and host metabolism. These results provide a theoretical basis for POP as a potential functional component against obesity.
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Registered trials
Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.