ReviewMolecular biology reports2026
microRNA-155: a versatile noncoding RNA.
Review in Molecular biology reports, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.
What it found
Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.
The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
The trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.
Who cites it
0 citing papers in PubMed.
No citing paper in PubMed yet.
Corrections and comments
PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.
Authors and funding
3 authors.
Funding
No grant is acknowledged in the PubMed record.
Abstract
MicroRNAs (miRNAs) are ~ 22-nucleotide-long noncoding RNAs that regulate gene expression at the post-transcriptional level through mRNA cleavage or translational repression. To date, approximately 2,700 mature human miRNAs have been identified, although biological functions have been assigned to fewer than 600. Among these, miR-155 has emerged as a regulatory miRNA with important roles in metabolism, immune homeostasis, and disease pathogenesis. Dysregulated miR-155 expression has been implicated in metabolic disorders, including diabetes and preeclampsia, as well as autoimmune diseases such as rheumatoid arthritis, multiple sclerosis, and systemic lupus erythematosus. Mechanistic and preclinical evidence indicates that aberrant miRNA-155 expression contributes to tumorigenesis by modulating tumor suppressor genes in hematologic malignancies and solid tumors. This review summarizes the current updates on the physiological and pathological roles of miRNA-155.
Indexed as
Identifiers
41860670What Socratic holds
Registered trials
Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.